- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-994X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Enhancer variants: evaluating functions in common disease ; volume:6 ; number:10 ; day:28 ; month:10 ; year:2014 ; pages:1-14 ; date:10.2014
Genome medicine ; 6, Heft 10 (28.10.2014), 1-14, 10.2014
- Creator
-
Corradin, Olivia
- Contributor
-
Scacheri, Peter C.
SpringerLink (Online service)
- DOI
-
10.1186/s13073-014-0085-3
- URN
-
urn:nbn:de:1111-201604099078
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:58 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Corradin, Olivia
- Scacheri, Peter C.
- SpringerLink (Online service)
Other Objects (12)
Affinity-optimizing enhancer variants disrupt development
A curated benchmark of enhancer-gene interactions for evaluating enhancer-target gene prediction methods
Enhancer RNAs: mechanisms in transcriptional regulation and functions in diseases
Evaluating functions to describe point patterns
DNA-binding factor footprints and enhancer RNAs identify functional non-coding genetic variants
Divergent chromatin accessibility dynamics promote differentiated enhancer functions and morphological trait evolution
Promiscuity of enhancer, coding and non-coding transcription functions in ultraconserved elements
Evolutionary relevance of single nucleotide variants within the forebrain exclusive human accelerated enhancer regions
Enhancer variants associated with Alzheimer’s disease affect gene expression via chromatin looping
Putative enhancer sites in the bovine genome are enriched with variants affecting complex traits
Enhancer variants reveal a conserved transcription factor network governed by PU.1 during osteoclast differentiation
The first enhancer in an enhancer chain safeguards subsequent enhancer-promoter contacts from a distance
Affinity-optimizing enhancer variants disrupt development
A curated benchmark of enhancer-gene interactions for evaluating enhancer-target gene prediction methods
Enhancer RNAs: mechanisms in transcriptional regulation and functions in diseases
Evaluating functions to describe point patterns
DNA-binding factor footprints and enhancer RNAs identify functional non-coding genetic variants
Divergent chromatin accessibility dynamics promote differentiated enhancer functions and morphological trait evolution
Promiscuity of enhancer, coding and non-coding transcription functions in ultraconserved elements
Evolutionary relevance of single nucleotide variants within the forebrain exclusive human accelerated enhancer regions
Enhancer variants associated with Alzheimer’s disease affect gene expression via chromatin looping
Putative enhancer sites in the bovine genome are enriched with variants affecting complex traits
Enhancer variants reveal a conserved transcription factor network governed by PU.1 during osteoclast differentiation
The first enhancer in an enhancer chain safeguards subsequent enhancer-promoter contacts from a distance
Affinity-optimizing enhancer variants disrupt development
A curated benchmark of enhancer-gene interactions for evaluating enhancer-target gene prediction methods
Enhancer RNAs: mechanisms in transcriptional regulation and functions in diseases
Evaluating functions to describe point patterns
DNA-binding factor footprints and enhancer RNAs identify functional non-coding genetic variants
Divergent chromatin accessibility dynamics promote differentiated enhancer functions and morphological trait evolution
Promiscuity of enhancer, coding and non-coding transcription functions in ultraconserved elements
Evolutionary relevance of single nucleotide variants within the forebrain exclusive human accelerated enhancer regions
Enhancer variants associated with Alzheimer’s disease affect gene expression via chromatin looping
Putative enhancer sites in the bovine genome are enriched with variants affecting complex traits
Enhancer variants reveal a conserved transcription factor network governed by PU.1 during osteoclast differentiation