Lactate inhibits the pro-inflammatory response and metabolic reprogramming in murine macrophages in a GPR81-independent manner
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
In: PLoS One, volume 11, issue 11, e0163694, doi:10.1371/journal.pone.0163694
- Keyword
-
Immunologie
Immunsystem
Lymphozyt
Leukozyt
Immunität
Resistenz
Immunchemie
- Event
-
Veröffentlichung
- (where)
-
Frankfurt am Main
- (who)
-
Universitätsbibliothek Johann Christian Senckenberg
- (when)
-
2016
- Creator
-
Errea, Agustina
Cayet, Delphine
Marchetti, Philippe
Tang, Cong
Kluza, Jerome
Offermanns, Stefan
Sirard, Jean-Claude
Rumbo, Martin
- URN
-
urn:nbn:de:hebis:30:3-419969
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:45 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Errea, Agustina
- Cayet, Delphine
- Marchetti, Philippe
- Tang, Cong
- Kluza, Jerome
- Offermanns, Stefan
- Sirard, Jean-Claude
- Rumbo, Martin
- Universitätsbibliothek Johann Christian Senckenberg
Time of origin
- 2016
Other Objects (12)
Reprogramming of pro-inflammatory human macrophages to an anti-inflammatory phenotype by bile acids
Glucocorticoid-induced immune-metabolic reprogramming in macrophages
Autophagic reprogramming of bone marrow–derived macrophages
Pharmacological reprogramming of macrophages through aptamiRs
Epigenetic reprogramming of macrophages in chronic diseases
PCSK9 induces a pro-inflammatory response in macrophages
Metabolic and inflammatory reprogramming of macrophages by ONC201 translates in a pro-inflammatory environment even in presence of glioblastoma cells
Metabolic and inflammatory reprogramming of macrophages by ONC201 translates in a pro‐inflammatory environment even in presence of glioblastoma cells
Reduction of Pro‐Inflammatory Markers in RAW264.7 Macrophages by Polyethylenimines
Dynamic reciprocal interactions between activated T cells and tumor associated macrophages drive macrophage reprogramming and proinflammatory T cell migration within prostate tumor models
Lactate oxidation facilitates growth of Mycobacterium tuberculosis in human macrophages
Metabolic reprogramming of macrophages and its involvement in inflammatory diseases
Reprogramming of pro-inflammatory human macrophages to an anti-inflammatory phenotype by bile acids
Glucocorticoid-induced immune-metabolic reprogramming in macrophages
Autophagic reprogramming of bone marrow–derived macrophages
Pharmacological reprogramming of macrophages through aptamiRs
Epigenetic reprogramming of macrophages in chronic diseases
PCSK9 induces a pro-inflammatory response in macrophages
Metabolic and inflammatory reprogramming of macrophages by ONC201 translates in a pro-inflammatory environment even in presence of glioblastoma cells
Metabolic and inflammatory reprogramming of macrophages by ONC201 translates in a pro‐inflammatory environment even in presence of glioblastoma cells
Reduction of Pro‐Inflammatory Markers in RAW264.7 Macrophages by Polyethylenimines
Dynamic reciprocal interactions between activated T cells and tumor associated macrophages drive macrophage reprogramming and proinflammatory T cell migration within prostate tumor models
Lactate oxidation facilitates growth of Mycobacterium tuberculosis in human macrophages
Metabolic reprogramming of macrophages and its involvement in inflammatory diseases
Reprogramming of pro-inflammatory human macrophages to an anti-inflammatory phenotype by bile acids
Glucocorticoid-induced immune-metabolic reprogramming in macrophages
Autophagic reprogramming of bone marrow–derived macrophages
Pharmacological reprogramming of macrophages through aptamiRs
Epigenetic reprogramming of macrophages in chronic diseases
PCSK9 induces a pro-inflammatory response in macrophages
Metabolic and inflammatory reprogramming of macrophages by ONC201 translates in a pro-inflammatory environment even in presence of glioblastoma cells
Metabolic and inflammatory reprogramming of macrophages by ONC201 translates in a pro‐inflammatory environment even in presence of glioblastoma cells
Reduction of Pro‐Inflammatory Markers in RAW264.7 Macrophages by Polyethylenimines
Dynamic reciprocal interactions between activated T cells and tumor associated macrophages drive macrophage reprogramming and proinflammatory T cell migration within prostate tumor models
Lactate oxidation facilitates growth of Mycobacterium tuberculosis in human macrophages