40 Hz light preserves synaptic plasticity and mitochondrial function in Alzheimer’s disease model
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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40 Hz light preserves synaptic plasticity and mitochondrial function in Alzheimer’s disease model ; volume:14 ; number:1 ; day:6 ; month:11 ; year:2024 ; pages:1-19 ; date:12.2024
Scientific reports ; 14, Heft 1 (6.11.2024), 1-19, 12.2024
- Creator
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Barzegar Behrooz, Amir
Aghanoori, Mohamad-Reza
Nazari, Maryam
Latifi-Navid, Hamid
Vosoughian, Fatemeh
Anjomani, Mojdeh
Lotfi, Jabar
Ahmadiani, Abolhassan
Eliassi, Afsaneh
Nabavizadeh, Fatemeh
Soleimani, Elham
Ghavami, Saeid
Khodagholi, Fariba
Fahanik-Babaei, Javad
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-024-78528-7
- URN
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urn:nbn:de:101:1-2501242117166.223230227851
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:28 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Barzegar Behrooz, Amir
- Aghanoori, Mohamad-Reza
- Nazari, Maryam
- Latifi-Navid, Hamid
- Vosoughian, Fatemeh
- Anjomani, Mojdeh
- Lotfi, Jabar
- Ahmadiani, Abolhassan
- Eliassi, Afsaneh
- Nabavizadeh, Fatemeh
- Soleimani, Elham
- Ghavami, Saeid
- Khodagholi, Fariba
- Fahanik-Babaei, Javad
- SpringerLink (Online service)
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Role of BACE1 in Alzheimer’s synaptic function
The benzodiazepine diazepam impairs synaptic plasticity via translocator protein (18 kDa): implications for Alzheimer’s disease
Oligomeric Aβ-induced synaptic dysfunction in Alzheimer’s disease
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High plasticity of axonal pathology in Alzheimer’s disease mouse models
Correction to: Alzheimer’s genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner
APP and Its Fragments in Synaptic Plasticity : Merging Physiological Insights with Alzheimer's and Tauopathy Research
Microbiota in neuroinflammation and synaptic dysfunction: a focus on Alzheimer’s disease
The role of pathological tau in synaptic dysfunction in Alzheimer’s diseases
Terminal complement pathway activation drives synaptic loss in Alzheimer’s disease models
Activity-homeostasis preserves synaptic plasticity in Purkinje cell but calcium is not the activity-sensor
Synaptic plasticity and the mechanism of Alzheimer's disease
Role of BACE1 in Alzheimer’s synaptic function
The benzodiazepine diazepam impairs synaptic plasticity via translocator protein (18 kDa): implications for Alzheimer’s disease
Oligomeric Aβ-induced synaptic dysfunction in Alzheimer’s disease
Alzheimer's disease: synaptic dysfunction and Aβ
High plasticity of axonal pathology in Alzheimer’s disease mouse models
Correction to: Alzheimer’s genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner
APP and Its Fragments in Synaptic Plasticity : Merging Physiological Insights with Alzheimer's and Tauopathy Research
Microbiota in neuroinflammation and synaptic dysfunction: a focus on Alzheimer’s disease
The role of pathological tau in synaptic dysfunction in Alzheimer’s diseases
Terminal complement pathway activation drives synaptic loss in Alzheimer’s disease models