Nuclear Localization Leucine-Rich-Repeat Protein 1 Deficiency Protects Against Cardiac Hypertrophy by Pressure Overload

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: Nuclear Localization Leucine-Rich-Repeat Protein 1 Deficiency Protects Against Cardiac Hypertrophy by Pressure Overload ; volume:48 ; number:1 ; year:2018 ; pages:75-86
Cellular physiology and biochemistry ; 48, Heft 1 (2018), 75-86

Creator: Zong, Jing
Li, Fang-fang
Liang, Kai
Dai, Rui
Zhang, Hao
Yan, Ling
Liu, Jia-li
Xu, Lu-hong
Qian, Wen-hao

DOI: 10.1159/000491664

URN: urn:nbn:de:101:1-2018083000032441883769

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:29 AM CEST

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Zong, Jing
Li, Fang-fang
Liang, Kai
Dai, Rui
Zhang, Hao
Yan, Ling
Liu, Jia-li
Xu, Lu-hong
Qian, Wen-hao

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Nuclear Localization Leucine-Rich-Repeat Protein 1 Deficiency Protects Against Cardiac Hypertrophy by Pressure Overload

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Other Objects (12)

2-Methoxyestradiol protects against pressure overload-induced left ventricular hypertrophy

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Functional characterization of leucine-rich repeat kinase 2 (LRRK2) dimerization

Nobiletin, a Polymethoxy Flavonoid, Protects Against Cardiac Hypertrophy Induced by Pressure-Overload via Inhibition of NAPDH Oxidases and Endoplasmic Reticulum Stress

Cellular hypertrophy occurs before interstitial fibrosis in pressure-overload heart failure

A cell surface interaction network of neural leucine-rich repeat receptors

Leucine-rich repeat kinase 1: Charakterisierung einer intramolekular GTP-regulierten Proteinkinase

Leucine-Rich repeat receptor kinases are sporadically distributed in eukaryotic genomes

CYP2J2 Overexpression Protects against Arrhythmia Susceptibility in Cardiac Hypertrophy

TNIP3 protects against pathological cardiac hypertrophy by stabilizing STAT1

NADPH oxidase-4 promotes eccentric cardiac hypertrophy in response to volume overload

2-Methoxyestradiol protects against pressure overload-induced left ventricular hypertrophy

CLERC and centrosomal leucine-rich repeat proteins

Interstitial expansion in pressure overload left ventricular hypertrophy

Functional characterization of leucine-rich repeat kinase 2 (LRRK2) dimerization

Nobiletin, a Polymethoxy Flavonoid, Protects Against Cardiac Hypertrophy Induced by Pressure-Overload via Inhibition of NAPDH Oxidases and Endoplasmic Reticulum Stress

Cellular hypertrophy occurs before interstitial fibrosis in pressure-overload heart failure

A cell surface interaction network of neural leucine-rich repeat receptors

Leucine-rich repeat kinase 1: Charakterisierung einer intramolekular GTP-regulierten Proteinkinase

Leucine-Rich repeat receptor kinases are sporadically distributed in eukaryotic genomes

CYP2J2 Overexpression Protects against Arrhythmia Susceptibility in Cardiac Hypertrophy

TNIP3 protects against pathological cardiac hypertrophy by stabilizing STAT1

NADPH oxidase-4 promotes eccentric cardiac hypertrophy in response to volume overload

2-Methoxyestradiol protects against pressure overload-induced left ventricular hypertrophy

CLERC and centrosomal leucine-rich repeat proteins

Interstitial expansion in pressure overload left ventricular hypertrophy

Functional characterization of leucine-rich repeat kinase 2 (LRRK2) dimerization

Nobiletin, a Polymethoxy Flavonoid, Protects Against Cardiac Hypertrophy Induced by Pressure-Overload via Inhibition of NAPDH Oxidases and Endoplasmic Reticulum Stress

Cellular hypertrophy occurs before interstitial fibrosis in pressure-overload heart failure

A cell surface interaction network of neural leucine-rich repeat receptors

Leucine-rich repeat kinase 1: Charakterisierung einer intramolekular GTP-regulierten Proteinkinase

Leucine-Rich repeat receptor kinases are sporadically distributed in eukaryotic genomes

CYP2J2 Overexpression Protects against Arrhythmia Susceptibility in Cardiac Hypertrophy

TNIP3 protects against pathological cardiac hypertrophy by stabilizing STAT1

NADPH oxidase-4 promotes eccentric cardiac hypertrophy in response to volume overload

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