- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1472-6831
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Sodium channel Nav1.8 immunoreactivity in painful human dental pulp ; volume:5 ; number:1 ; day:07 ; month:7 ; year:2005 ; pages:1-6 ; date:12.2005
BMC oral health ; 5, Heft 1 (07.7.2005), 1-6, 12.2005
- Urheber
-
Renton, T.
- Beteiligte Personen und Organisationen
-
Yiangou, Y.
Plumpton, C.
Tate, S.
Bountra, C.
Anand, P.
SpringerLink (Online service)
- DOI
-
10.1186/1472-6831-5-5
- URN
-
urn:nbn:de:1111-2016071630958
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 11:00 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Renton, T.
- Yiangou, Y.
- Plumpton, C.
- Tate, S.
- Bountra, C.
- Anand, P.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Sodium channel Nav1.7 immunoreactivity in painful human dental pulp and burning mouth syndrome
Network topology of NaV1.7 mutations in sodium channel-related painful disorders
Nav1.7 expression is increased in painful human dental pulp
Unmyelinated nerve fibers in the human dental pulp express markers for myelinated fibers and show sodium channel accumulations
Sodium channel slow inactivation interferes with open channel block
Sodium channel biophysics, late sodium current and genetic arrhythmic syndromes
Painful and painless mutations of SCN9A and SCN11A voltage-gated sodium channels
Reduction of voltage gated sodium channel protein in DRG by vector mediated miRNA reduces pain in rats with painful diabetic neuropathy
Painful Peace
Pharmacological modification of sodium channel Nav1.9 activity
Macrophages enhance sodium channel expression in cardiomyocytes
Cav3.2 calcium channel interactions with the epithelial sodium channel ENaC
Sodium channel Nav1.7 immunoreactivity in painful human dental pulp and burning mouth syndrome
Network topology of NaV1.7 mutations in sodium channel-related painful disorders
Nav1.7 expression is increased in painful human dental pulp
Unmyelinated nerve fibers in the human dental pulp express markers for myelinated fibers and show sodium channel accumulations
Sodium channel slow inactivation interferes with open channel block
Sodium channel biophysics, late sodium current and genetic arrhythmic syndromes
Painful and painless mutations of SCN9A and SCN11A voltage-gated sodium channels
Reduction of voltage gated sodium channel protein in DRG by vector mediated miRNA reduces pain in rats with painful diabetic neuropathy
Painful Peace
Pharmacological modification of sodium channel Nav1.9 activity
Macrophages enhance sodium channel expression in cardiomyocytes
Cav3.2 calcium channel interactions with the epithelial sodium channel ENaC
Sodium channel Nav1.7 immunoreactivity in painful human dental pulp and burning mouth syndrome
Network topology of NaV1.7 mutations in sodium channel-related painful disorders
Nav1.7 expression is increased in painful human dental pulp
Unmyelinated nerve fibers in the human dental pulp express markers for myelinated fibers and show sodium channel accumulations
Sodium channel slow inactivation interferes with open channel block
Sodium channel biophysics, late sodium current and genetic arrhythmic syndromes
Painful and painless mutations of SCN9A and SCN11A voltage-gated sodium channels
Reduction of voltage gated sodium channel protein in DRG by vector mediated miRNA reduces pain in rats with painful diabetic neuropathy
Painful Peace
Pharmacological modification of sodium channel Nav1.9 activity
Macrophages enhance sodium channel expression in cardiomyocytes