Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2041-4889

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a ; volume:13 ; number:8 ; day:12 ; month:8 ; year:2022 ; pages:1-13 ; date:8.2022
Cell death & disease ; 13, Heft 8 (12.8.2022), 1-13, 8.2022

Creator: Clusmann, Jan Niklas
Franco, Klaus-Daniel Cortés
Suárez, David Alejandro Corredor
Katona, Istvan
Minguez, Maria Girbes
Boersch, Nina
Pissas, Karolos-Philippos
Vanek, Jakob
Tian, Yuemin
Gründer, Stefan

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41419-022-05139-3

URN: urn:nbn:de:101:1-2022112709504582141665

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:36 AM CEST

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Associated

Clusmann, Jan Niklas
Franco, Klaus-Daniel Cortés
Suárez, David Alejandro Corredor
Katona, Istvan
Minguez, Maria Girbes
Boersch, Nina
Pissas, Karolos-Philippos
Vanek, Jakob
Tian, Yuemin
Gründer, Stefan
SpringerLink (Online service)

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Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

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Short-Term Differentiation of Glioblastoma Stem Cells Induces Hypoxia Tolerance

Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

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Acid-sensing ion channel 1a induziert Zelltod in Glioblastomstammzellen via RIPK1

Human ZBP1 induces cell death‐independent inflammatory signaling via RIPK3 and RIPK1

Acidosis

Targeting the Potassium Channel Kv1.3 Kills Glioblastoma Cells

The structure of mouse RIPK1 RHIM-containing domain as a homo-amyloid and in RIPK1/RIPK3 complex

Acidosis-induced activation of anion channel SLAH3 in the flooding-related stress response of Arabidopsis

Progesterone Treatment Attenuates Glycolytic Metabolism and Induces Senescence in Glioblastoma

IGFBP3 induces PD-L1 expression to promote glioblastoma immune evasion

Knockdown of ribosomal protein S15A induces human glioblastoma cell apoptosis

Osimertinib induces paraptosis and TRIP13 confers resistance in glioblastoma cells

Short-Term Differentiation of Glioblastoma Stem Cells Induces Hypoxia Tolerance

Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

Hochschulschrift

Acid-sensing ion channel 1a induziert Zelltod in Glioblastomstammzellen via RIPK1

Human ZBP1 induces cell death‐independent inflammatory signaling via RIPK3 and RIPK1

Acidosis

Targeting the Potassium Channel Kv1.3 Kills Glioblastoma Cells

The structure of mouse RIPK1 RHIM-containing domain as a homo-amyloid and in RIPK1/RIPK3 complex

Acidosis-induced activation of anion channel SLAH3 in the flooding-related stress response of Arabidopsis

Progesterone Treatment Attenuates Glycolytic Metabolism and Induces Senescence in Glioblastoma

IGFBP3 induces PD-L1 expression to promote glioblastoma immune evasion

Knockdown of ribosomal protein S15A induces human glioblastoma cell apoptosis

Osimertinib induces paraptosis and TRIP13 confers resistance in glioblastoma cells

Short-Term Differentiation of Glioblastoma Stem Cells Induces Hypoxia Tolerance

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Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

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Other Objects (12)

Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

Acid-sensing ion channel 1a induziert Zelltod in Glioblastomstammzellen via RIPK1

Human ZBP1 induces cell death‐independent inflammatory signaling via RIPK3 and RIPK1

Acidosis

Targeting the Potassium Channel Kv1.3 Kills Glioblastoma Cells

The structure of mouse RIPK1 RHIM-containing domain as a homo-amyloid and in RIPK1/RIPK3 complex

Acidosis-induced activation of anion channel SLAH3 in the flooding-related stress response of Arabidopsis

Progesterone Treatment Attenuates Glycolytic Metabolism and Induces Senescence in Glioblastoma

IGFBP3 induces PD-L1 expression to promote glioblastoma immune evasion

Knockdown of ribosomal protein S15A induces human glioblastoma cell apoptosis

Osimertinib induces paraptosis and TRIP13 confers resistance in glioblastoma cells

Short-Term Differentiation of Glioblastoma Stem Cells Induces Hypoxia Tolerance

Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

Acid-sensing ion channel 1a induziert Zelltod in Glioblastomstammzellen via RIPK1

Human ZBP1 induces cell death‐independent inflammatory signaling via RIPK3 and RIPK1

Acidosis

Targeting the Potassium Channel Kv1.3 Kills Glioblastoma Cells

The structure of mouse RIPK1 RHIM-containing domain as a homo-amyloid and in RIPK1/RIPK3 complex

Acidosis-induced activation of anion channel SLAH3 in the flooding-related stress response of Arabidopsis

Progesterone Treatment Attenuates Glycolytic Metabolism and Induces Senescence in Glioblastoma

IGFBP3 induces PD-L1 expression to promote glioblastoma immune evasion

Knockdown of ribosomal protein S15A induces human glioblastoma cell apoptosis

Osimertinib induces paraptosis and TRIP13 confers resistance in glioblastoma cells

Short-Term Differentiation of Glioblastoma Stem Cells Induces Hypoxia Tolerance

Acidosis induces RIPK1-dependent death of glioblastoma stem cells via acid-sensing ion channel 1a

Acid-sensing ion channel 1a induziert Zelltod in Glioblastomstammzellen via RIPK1

Human ZBP1 induces cell death‐independent inflammatory signaling via RIPK3 and RIPK1

Acidosis

Targeting the Potassium Channel Kv1.3 Kills Glioblastoma Cells

The structure of mouse RIPK1 RHIM-containing domain as a homo-amyloid and in RIPK1/RIPK3 complex

Acidosis-induced activation of anion channel SLAH3 in the flooding-related stress response of Arabidopsis

Progesterone Treatment Attenuates Glycolytic Metabolism and Induces Senescence in Glioblastoma

IGFBP3 induces PD-L1 expression to promote glioblastoma immune evasion

Knockdown of ribosomal protein S15A induces human glioblastoma cell apoptosis

Osimertinib induces paraptosis and TRIP13 confers resistance in glioblastoma cells

Short-Term Differentiation of Glioblastoma Stem Cells Induces Hypoxia Tolerance

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