Itch induced by peripheral mu opioid receptors is dependent on TRPV1-expressing neurons and alleviated by channel activation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Itch induced by peripheral mu opioid receptors is dependent on TRPV1-expressing neurons and alleviated by channel activation ; volume:8 ; number:1 ; day:19 ; month:10 ; year:2018 ; pages:1-9 ; date:12.2018
Scientific reports ; 8, Heft 1 (19.10.2018), 1-9, 12.2018
- Creator
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Melo, Helvira
- Contributor
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Basso, Lilian
Iftinca, Mircea
MacNaughton, Wallace K.
Hollenberg, Morley D.
McKay, Derek M.
Altier, Christophe
SpringerLink (Online service)
- DOI
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10.1038/s41598-018-33620-7
- URN
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urn:nbn:de:101:1-2018112100363949656382
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:50 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Melo, Helvira
- Basso, Lilian
- Iftinca, Mircea
- MacNaughton, Wallace K.
- Hollenberg, Morley D.
- McKay, Derek M.
- Altier, Christophe
- SpringerLink (Online service)
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Activation profiles of opioid ligands in HEK cells expressing δ opioid receptors
Molecular recognition of itch-associated neuropeptides by bombesin receptors
Activation of NPY2R-expressing amygdala neurons inhibits itch behavior in mice without lateralization
STING controls opioid-induced itch and chronic itch via spinal tank-binding kinase 1-dependent type I interferon response in mice
Understand spiciness: mechanism of TRPV1 channel activation by capsaicin
TLR4 enhances histamine-mediated pruritus by potentiating TRPV1 activity
Structural basis of TRPV1 modulation by endogenous bioactive lipids
AKAP150-mediated TRPV1 sensitization is disrupted by calcium/calmodulin
Structural basis of TRPV1 inhibition by SAF312 and cholesterol
Sophorolipid inhibits histamine-induced itch by decreasing PLC/IP3R signaling pathway activation and modulating TRPV1 activity
Stimulation of TRPV1 by green laser light
Chronic itch induced by thalamic deep brain stimulation: a case for a central itch centre
Activation profiles of opioid ligands in HEK cells expressing δ opioid receptors
Molecular recognition of itch-associated neuropeptides by bombesin receptors
Activation of NPY2R-expressing amygdala neurons inhibits itch behavior in mice without lateralization
STING controls opioid-induced itch and chronic itch via spinal tank-binding kinase 1-dependent type I interferon response in mice
Understand spiciness: mechanism of TRPV1 channel activation by capsaicin
TLR4 enhances histamine-mediated pruritus by potentiating TRPV1 activity
Structural basis of TRPV1 modulation by endogenous bioactive lipids
AKAP150-mediated TRPV1 sensitization is disrupted by calcium/calmodulin
Structural basis of TRPV1 inhibition by SAF312 and cholesterol
Sophorolipid inhibits histamine-induced itch by decreasing PLC/IP3R signaling pathway activation and modulating TRPV1 activity
Stimulation of TRPV1 by green laser light
Chronic itch induced by thalamic deep brain stimulation: a case for a central itch centre
Activation profiles of opioid ligands in HEK cells expressing δ opioid receptors
Molecular recognition of itch-associated neuropeptides by bombesin receptors
Activation of NPY2R-expressing amygdala neurons inhibits itch behavior in mice without lateralization
STING controls opioid-induced itch and chronic itch via spinal tank-binding kinase 1-dependent type I interferon response in mice
Understand spiciness: mechanism of TRPV1 channel activation by capsaicin
TLR4 enhances histamine-mediated pruritus by potentiating TRPV1 activity
Structural basis of TRPV1 modulation by endogenous bioactive lipids
AKAP150-mediated TRPV1 sensitization is disrupted by calcium/calmodulin