Activation of the integrated stress response induces remodeling of cardiac metabolism in Barth Syndrome

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Alternative title: Aktivierung der "Integrated Stress Response" führt zur Umstellung des kardialen Metabolismus im Barth Syndrom

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Notes: Würzburg, Universität Würzburg, Dissertation, 0024

Classification: Medizin, Gesundheit

Keyword: Herzmuskelkrankheit
Mitochondrium
Stoffwechsel

Event: Veröffentlichung

(where): Würzburg

(who): Universität Würzburg

(when): 2024

Creator: Kutschka, Ilona

Contributor: Maack, Christoph
Schulze, Almut

DOI: 10.25972/OPUS-35818

URN: urn:nbn:de:bvb:20-opus-358186

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:45 AM CEST

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Associated

Kutschka, Ilona
Maack, Christoph
Schulze, Almut
Universität Würzburg

Time of origin

2024

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TEA domain transcription factor 1 (TEAD1) induces cardiac fibroblasts cells remodeling through BRD4/Wnt4 pathway

Neurohormonal activation induces intracellular iron deficiency and mitochondrial dysfunction in cardiac cells

Phosphorylation of the chromatin remodeling factor DPF3a induces cardiac hypertrophy through releasing HEY repressors from DNA

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Cardiac Remodeling in Chronic Kidney Disease

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Macrophage activation and polarization in post-infarction cardiac remodeling

Givinostat reduces adverse cardiac remodeling through regulating fibroblasts activation

Efavirenz induces alterations in lipid metabolism through AMPK activation

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Constitutive activation of FoxO3 induces autophagy and cardiac atrophy

Chromatin remodeling-driven autophagy activation induces cisplatin resistance in oral squamous cell carcinoma

Nuclear import of Mas-related G protein-coupled receptor member D induces pathological cardiac remodeling

TEA domain transcription factor 1 (TEAD1) induces cardiac fibroblasts cells remodeling through BRD4/Wnt4 pathway

Neurohormonal activation induces intracellular iron deficiency and mitochondrial dysfunction in cardiac cells

Phosphorylation of the chromatin remodeling factor DPF3a induces cardiac hypertrophy through releasing HEY repressors from DNA

Aufsatzsammlung

Cardiac vascular remodeling and functional interaction

Cardiac Remodeling in Chronic Kidney Disease

Cardiac conduction velocity, remodeling and arrhythmogenesis

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Activation of the integrated stress response induces remodeling of cardiac metabolism in Barth Syndrome

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Other Objects (12)

Macrophage activation and polarization in post-infarction cardiac remodeling

Givinostat reduces adverse cardiac remodeling through regulating fibroblasts activation

Efavirenz induces alterations in lipid metabolism through AMPK activation

Constitutive activation of FoxO3 induces autophagy and cardiac atrophy

Chromatin remodeling-driven autophagy activation induces cisplatin resistance in oral squamous cell carcinoma

Nuclear import of Mas-related G protein-coupled receptor member D induces pathological cardiac remodeling

TEA domain transcription factor 1 (TEAD1) induces cardiac fibroblasts cells remodeling through BRD4/Wnt4 pathway

Neurohormonal activation induces intracellular iron deficiency and mitochondrial dysfunction in cardiac cells

Phosphorylation of the chromatin remodeling factor DPF3a induces cardiac hypertrophy through releasing HEY repressors from DNA

Cardiac vascular remodeling and functional interaction

Cardiac Remodeling in Chronic Kidney Disease

Cardiac conduction velocity, remodeling and arrhythmogenesis

Macrophage activation and polarization in post-infarction cardiac remodeling

Givinostat reduces adverse cardiac remodeling through regulating fibroblasts activation

Efavirenz induces alterations in lipid metabolism through AMPK activation

Constitutive activation of FoxO3 induces autophagy and cardiac atrophy

Chromatin remodeling-driven autophagy activation induces cisplatin resistance in oral squamous cell carcinoma

Nuclear import of Mas-related G protein-coupled receptor member D induces pathological cardiac remodeling

TEA domain transcription factor 1 (TEAD1) induces cardiac fibroblasts cells remodeling through BRD4/Wnt4 pathway

Neurohormonal activation induces intracellular iron deficiency and mitochondrial dysfunction in cardiac cells

Phosphorylation of the chromatin remodeling factor DPF3a induces cardiac hypertrophy through releasing HEY repressors from DNA

Cardiac vascular remodeling and functional interaction

Cardiac Remodeling in Chronic Kidney Disease

Cardiac conduction velocity, remodeling and arrhythmogenesis

Macrophage activation and polarization in post-infarction cardiac remodeling

Givinostat reduces adverse cardiac remodeling through regulating fibroblasts activation

Efavirenz induces alterations in lipid metabolism through AMPK activation

Constitutive activation of FoxO3 induces autophagy and cardiac atrophy

Chromatin remodeling-driven autophagy activation induces cisplatin resistance in oral squamous cell carcinoma

Nuclear import of Mas-related G protein-coupled receptor member D induces pathological cardiac remodeling

TEA domain transcription factor 1 (TEAD1) induces cardiac fibroblasts cells remodeling through BRD4/Wnt4 pathway

Neurohormonal activation induces intracellular iron deficiency and mitochondrial dysfunction in cardiac cells

Phosphorylation of the chromatin remodeling factor DPF3a induces cardiac hypertrophy through releasing HEY repressors from DNA

Cardiac vascular remodeling and functional interaction

Cardiac Remodeling in Chronic Kidney Disease

Cardiac conduction velocity, remodeling and arrhythmogenesis

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