Selective memory and behavioral alterations after ambient ultrafine particulate matter exposure in aged 3xTgAD Alzheimer’s disease mice
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1743-8977
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Selective memory and behavioral alterations after ambient ultrafine particulate matter exposure in aged 3xTgAD Alzheimer’s disease mice ; volume:16 ; number:1 ; day:26 ; month:11 ; year:2019 ; pages:1-17 ; date:12.2019
Particle and fibre toxicology ; 16, Heft 1 (26.11.2019), 1-17, 12.2019
- Urheber
-
Jew, Katrina
Herr, Denise
Wong, Candace
Kennell, Andrea
Morris-Schaffer, Keith
Oberdörster, Günter
O'Banion, M. Kerry
Cory-Slechta, Deborah A.
Elder, Alison
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s12989-019-0323-3
- URN
-
urn:nbn:de:101:1-2020032119043877537420
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:48 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Jew, Katrina
- Herr, Denise
- Wong, Candace
- Kennell, Andrea
- Morris-Schaffer, Keith
- Oberdörster, Günter
- O'Banion, M. Kerry
- Cory-Slechta, Deborah A.
- Elder, Alison
- SpringerLink (Online service)
Ähnliche Objekte (12)
Impaired Spatial Reorientation in the 3xTg-AD Mouse Model of Alzheimer’s Disease
Enhanced susceptibility of triple transgenic Alzheimer’s disease (3xTg-AD) mice to acute infection
A multi-targeting immunotherapy ameliorates multiple facets of Alzheimer’s disease in 3xTg mice
Short-term resistance exercise inhibits neuroinflammation and attenuates neuropathological changes in 3xTg Alzheimer’s disease mice
Exercise and resveratrol increase fracture resistance in the 3xTg-AD mouse model of Alzheimer’s disease
Fingolimod Rescues Memory and Improves Pathological Hallmarks in the 3xTg-AD Model of Alzheimer’s Disease
Neuroprotective effects of bee venom phospholipase A2 in the 3xTg AD mouse model of Alzheimer’s disease
IVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathology
Immunomodulation targeting of both Aβ and tau pathological conformers ameliorates Alzheimer’s disease pathology in TgSwDI and 3xTg mouse models
Sex differences in metabolic phenotype and hypothalamic inflammation in the 3xTg-AD mouse model of Alzheimer’s disease
Differential effects of chronic immunosuppression on behavioral, epigenetic, and Alzheimer’s disease-associated markers in 3xTg-AD mice
Abundance of Aβ5-xlike immunoreactivity in transgenic 5XFAD, APP/PS1KI and 3xTG mice, sporadic and familial Alzheimer’s disease
Impaired Spatial Reorientation in the 3xTg-AD Mouse Model of Alzheimer’s Disease
Enhanced susceptibility of triple transgenic Alzheimer’s disease (3xTg-AD) mice to acute infection
A multi-targeting immunotherapy ameliorates multiple facets of Alzheimer’s disease in 3xTg mice
Short-term resistance exercise inhibits neuroinflammation and attenuates neuropathological changes in 3xTg Alzheimer’s disease mice
Exercise and resveratrol increase fracture resistance in the 3xTg-AD mouse model of Alzheimer’s disease
Fingolimod Rescues Memory and Improves Pathological Hallmarks in the 3xTg-AD Model of Alzheimer’s Disease
Neuroprotective effects of bee venom phospholipase A2 in the 3xTg AD mouse model of Alzheimer’s disease
IVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathology
Immunomodulation targeting of both Aβ and tau pathological conformers ameliorates Alzheimer’s disease pathology in TgSwDI and 3xTg mouse models
Sex differences in metabolic phenotype and hypothalamic inflammation in the 3xTg-AD mouse model of Alzheimer’s disease
Differential effects of chronic immunosuppression on behavioral, epigenetic, and Alzheimer’s disease-associated markers in 3xTg-AD mice
Abundance of Aβ5-xlike immunoreactivity in transgenic 5XFAD, APP/PS1KI and 3xTG mice, sporadic and familial Alzheimer’s disease
Impaired Spatial Reorientation in the 3xTg-AD Mouse Model of Alzheimer’s Disease
Enhanced susceptibility of triple transgenic Alzheimer’s disease (3xTg-AD) mice to acute infection
A multi-targeting immunotherapy ameliorates multiple facets of Alzheimer’s disease in 3xTg mice
Short-term resistance exercise inhibits neuroinflammation and attenuates neuropathological changes in 3xTg Alzheimer’s disease mice
Exercise and resveratrol increase fracture resistance in the 3xTg-AD mouse model of Alzheimer’s disease
Fingolimod Rescues Memory and Improves Pathological Hallmarks in the 3xTg-AD Model of Alzheimer’s Disease
Neuroprotective effects of bee venom phospholipase A2 in the 3xTg AD mouse model of Alzheimer’s disease
IVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathology
Immunomodulation targeting of both Aβ and tau pathological conformers ameliorates Alzheimer’s disease pathology in TgSwDI and 3xTg mouse models
Sex differences in metabolic phenotype and hypothalamic inflammation in the 3xTg-AD mouse model of Alzheimer’s disease
Differential effects of chronic immunosuppression on behavioral, epigenetic, and Alzheimer’s disease-associated markers in 3xTg-AD mice