Phase II study of talazoparib in advanced cancers with BRCA1/2, DNA repair, and PTEN alterations

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Phase II study of talazoparib in advanced cancers with BRCA1/2, DNA repair, and PTEN alterations ; volume:8 ; number:1 ; day:31 ; month:7 ; year:2024 ; pages:1-14 ; date:12.2024
npj precision oncology ; 8, Heft 1 (31.7.2024), 1-14, 12.2024

Creator: Piha-Paul, Sarina A.
Tseng, Chieh
Leung, Cheuk Hong
Yuan, Ying
Karp, Daniel D.
Subbiah, Vivek
Hong, David
Fu, Siqing
Naing, Aung
Rodon, Jordi
Javle, Milind
Ajani, Jaffer A.
Raghav, Kanwal P.
Somaiah, Neeta
Mills, Gordon B.
Tsimberidou, Apostolia M.
Zheng, Xiaofeng
Chen, Ken
Meric-Bernstam, Funda

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41698-024-00634-6

URN: urn:nbn:de:101:1-2410250843479.833437116246

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:24 AM CEST

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Piha-Paul, Sarina A.
Tseng, Chieh
Leung, Cheuk Hong
Yuan, Ying
Karp, Daniel D.
Subbiah, Vivek
Hong, David
Fu, Siqing
Naing, Aung
Rodon, Jordi
Javle, Milind
Ajani, Jaffer A.
Raghav, Kanwal P.
Somaiah, Neeta
Mills, Gordon B.
Tsimberidou, Apostolia M.
Zheng, Xiaofeng
Chen, Ken
Meric-Bernstam, Funda
SpringerLink (Online service)

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Phase II study of talazoparib in advanced cancers with BRCA1/2, DNA repair, and PTEN alterations

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Other Objects (12)

PARG deficiency is neither synthetic lethal with BRCA1 nor PTEN deficiency

Alterations in PTEN and PIK3CA in colorectal cancers in the EPIC Norfolk study: associations with clinicopathological and dietary factors

Modelling aggressive prostate cancers of young men in immune-competent mice, driven by isogenic Trp53 alterations and Pten loss

Prevalence of APC and PTEN Alterations in Urachal Cancer

Prevalence of APC and PTEN Alterations in Urachal Cancer

Cytotoxic and targeted therapy for BRCA1/2-driven cancers

The BRCA1/2 pathway prevents hematologic cancers in addition to breast and ovarian cancers

PTEN loss correlates with T cell exclusion across human cancers

PIK3CA alterations in Middle Eastern ovarian cancers

Genomic alterations in oral multiple primary cancers

SWI/SNF Alterations in Squamous Bladder Cancers

The dysregulation of BRCA1, PTEN, or CHK1 influences therapy resistance of breast cancer cells by affecting DNA repair and DNA replication stress signaling

PARG deficiency is neither synthetic lethal with BRCA1 nor PTEN deficiency

Alterations in PTEN and PIK3CA in colorectal cancers in the EPIC Norfolk study: associations with clinicopathological and dietary factors

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Prevalence of APC and PTEN Alterations in Urachal Cancer

Prevalence of APC and PTEN Alterations in Urachal Cancer

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The BRCA1/2 pathway prevents hematologic cancers in addition to breast and ovarian cancers

PTEN loss correlates with T cell exclusion across human cancers

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SWI/SNF Alterations in Squamous Bladder Cancers

The dysregulation of BRCA1, PTEN, or CHK1 influences therapy resistance of breast cancer cells by affecting DNA repair and DNA replication stress signaling

PARG deficiency is neither synthetic lethal with BRCA1 nor PTEN deficiency

Alterations in PTEN and PIK3CA in colorectal cancers in the EPIC Norfolk study: associations with clinicopathological and dietary factors

Modelling aggressive prostate cancers of young men in immune-competent mice, driven by isogenic Trp53 alterations and Pten loss

Prevalence of APC and PTEN Alterations in Urachal Cancer

Prevalence of APC and PTEN Alterations in Urachal Cancer

Cytotoxic and targeted therapy for BRCA1/2-driven cancers

The BRCA1/2 pathway prevents hematologic cancers in addition to breast and ovarian cancers

PTEN loss correlates with T cell exclusion across human cancers

PIK3CA alterations in Middle Eastern ovarian cancers

Genomic alterations in oral multiple primary cancers

SWI/SNF Alterations in Squamous Bladder Cancers

The dysregulation of BRCA1, PTEN, or CHK1 influences therapy resistance of breast cancer cells by affecting DNA repair and DNA replication stress signaling

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