Activation of NPY2R-expressing amygdala neurons inhibits itch behavior in mice without lateralization
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Activation of NPY2R-expressing amygdala neurons inhibits itch behavior in mice without lateralization ; volume:14 ; number:1 ; day:27 ; month:9 ; year:2024 ; pages:1-11 ; date:12.2024
Scientific reports ; 14, Heft 1 (27.9.2024), 1-11, 12.2024
- Creator
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Pavlenko, Darya
Todurga-Seven, Zeynep Gizem
Sanders, Kristen
Markan, Anika
Verpile, Rebecca
Ishida, Hirotake
Costo, Dylan
Akiyama, Tasuku
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-024-73483-9
- URN
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urn:nbn:de:101:1-2412132129465.447664534829
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:34 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Pavlenko, Darya
- Todurga-Seven, Zeynep Gizem
- Sanders, Kristen
- Markan, Anika
- Verpile, Rebecca
- Ishida, Hirotake
- Costo, Dylan
- Akiyama, Tasuku
- SpringerLink (Online service)
Other Objects (12)
Hemispheric lateralization of a molecular signal for pain modulation in the amygdala
Kidney Itch
itchY
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NR2B receptor blockade inhibits pain-related sensitization of amygdala neurons
Lateralization of the 5-HT1A receptors in the basolateral amygdala in metabolic and anxiety responses to chronic restraint stress
Attentional processing of itch
Role of Calcr expressing neurons in the medial amygdala in social contact among females
Basolateral amygdala parvalbumin and cholecystokinin-expressing GABAergic neurons modulate depressive and anxiety-like behaviors
Distraction From Itch Shows Brainstem Activation Without Reduction in Experimental Itch Sensation
Itch induced by peripheral mu opioid receptors is dependent on TRPV1-expressing neurons and alleviated by channel activation
Hemispheric lateralization of a molecular signal for pain modulation in the amygdala
Kidney Itch
itchY
Psychogenic itch
Pharmacology of itch
NR2B receptor blockade inhibits pain-related sensitization of amygdala neurons
Lateralization of the 5-HT1A receptors in the basolateral amygdala in metabolic and anxiety responses to chronic restraint stress
Attentional processing of itch
Role of Calcr expressing neurons in the medial amygdala in social contact among females
Basolateral amygdala parvalbumin and cholecystokinin-expressing GABAergic neurons modulate depressive and anxiety-like behaviors
Distraction From Itch Shows Brainstem Activation Without Reduction in Experimental Itch Sensation
Itch induced by peripheral mu opioid receptors is dependent on TRPV1-expressing neurons and alleviated by channel activation
Hemispheric lateralization of a molecular signal for pain modulation in the amygdala
Kidney Itch
itchY
Psychogenic itch
Pharmacology of itch
NR2B receptor blockade inhibits pain-related sensitization of amygdala neurons
Lateralization of the 5-HT1A receptors in the basolateral amygdala in metabolic and anxiety responses to chronic restraint stress
Attentional processing of itch
Role of Calcr expressing neurons in the medial amygdala in social contact among females
Basolateral amygdala parvalbumin and cholecystokinin-expressing GABAergic neurons modulate depressive and anxiety-like behaviors
Distraction From Itch Shows Brainstem Activation Without Reduction in Experimental Itch Sensation