The proteasome complex and the maintenance of pluripotency: sustain the fate by mopping up?
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1757-6512
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
The proteasome complex and the maintenance of pluripotency: sustain the fate by mopping up? ; volume:5 ; number:1 ; day:18 ; month:2 ; year:2014 ; pages:1-9 ; date:3.2014
Stem cell research & therapy ; 5, Heft 1 (18.2.2014), 1-9, 3.2014
- Urheber
-
Schröter, Friederike
- Beteiligte Personen und Organisationen
-
Adjaye, James
SpringerLink (Online service)
- DOI
-
10.1186/scrt413
- URN
-
urn:nbn:de:1111-2016073133940
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:58 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Schröter, Friederike
- Adjaye, James
- SpringerLink (Online service)
Ähnliche Objekte (12)
Tracing the transitions from pluripotency to germ cell fate with CRISPR screening
The fate of endoplasmic reticulum-targeted proteins in the face of proteasome failure
Switching Human Cell Fate: Reprogramming via pluripotency to adipogenesis
Publisher Correction: Tracing the transitions from pluripotency to germ cell fate with CRISPR screening
Membrane potential drives the exit from pluripotency and cell fate commitment via calcium and mTOR
Metabolic regulation of pluripotency and germ cell fate through α‐ketoglutarate
Genetic tool for fate mapping of Oct4 (Pou5f1)-expressing cells and their progeny past the pluripotency stage
Should we sustain? And if so, sustain what? Consumption or the quality of life?
Modeling signaling‐dependent pluripotency with Boolean logic to predict cell fate transitions
Taking the RISC of exiting naïve pluripotency
The role of Myc in the ground state of pluripotency
Molecular mechanisms involved in the induction of pluripotency
Tracing the transitions from pluripotency to germ cell fate with CRISPR screening
The fate of endoplasmic reticulum-targeted proteins in the face of proteasome failure
Switching Human Cell Fate: Reprogramming via pluripotency to adipogenesis
Publisher Correction: Tracing the transitions from pluripotency to germ cell fate with CRISPR screening
Membrane potential drives the exit from pluripotency and cell fate commitment via calcium and mTOR
Metabolic regulation of pluripotency and germ cell fate through α‐ketoglutarate
Genetic tool for fate mapping of Oct4 (Pou5f1)-expressing cells and their progeny past the pluripotency stage
Should we sustain? And if so, sustain what? Consumption or the quality of life?
Modeling signaling‐dependent pluripotency with Boolean logic to predict cell fate transitions
Taking the RISC of exiting naïve pluripotency
The role of Myc in the ground state of pluripotency
Molecular mechanisms involved in the induction of pluripotency
Tracing the transitions from pluripotency to germ cell fate with CRISPR screening
The fate of endoplasmic reticulum-targeted proteins in the face of proteasome failure
Switching Human Cell Fate: Reprogramming via pluripotency to adipogenesis
Publisher Correction: Tracing the transitions from pluripotency to germ cell fate with CRISPR screening
Membrane potential drives the exit from pluripotency and cell fate commitment via calcium and mTOR
Metabolic regulation of pluripotency and germ cell fate through α‐ketoglutarate
Genetic tool for fate mapping of Oct4 (Pou5f1)-expressing cells and their progeny past the pluripotency stage
Should we sustain? And if so, sustain what? Consumption or the quality of life?
Modeling signaling‐dependent pluripotency with Boolean logic to predict cell fate transitions
Taking the RISC of exiting naïve pluripotency
The role of Myc in the ground state of pluripotency