Cyclophosphamide-induced testicular injury: the role of chrysin in mitigating iron overload and ferroptosis
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Cyclophosphamide-induced testicular injury: the role of chrysin in mitigating iron overload and ferroptosis ; day:20 ; month:11 ; year:2024 ; pages:1-15
Naunyn-Schmiedeberg's archives of pharmacology ; (20.11.2024), 1-15
- Creator
-
Saleh, Dalia O.
Abo El Nasr, Nesma M. E.
Hussien, Yosra A.
El-Baset, Marawan Abd
Ahmed, Kawkab A.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s00210-024-03519-4
- URN
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urn:nbn:de:101:1-2502062129574.046795383012
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:25 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Saleh, Dalia O.
- Abo El Nasr, Nesma M. E.
- Hussien, Yosra A.
- El-Baset, Marawan Abd
- Ahmed, Kawkab A.
- SpringerLink (Online service)
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Cyclophosphamide induces ovarian granulosa cell ferroptosis via a mechanism associated with HO-1 and ROS-mediated mitochondrial dysfunction
Leptin-mediated downregulation of glutathione peroxidase 4 and iron overload contributing to podocyte ferroptosis in diabetic nephropathy
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PPARα alleviates iron overload‐induced ferroptosis in mouse liver
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ARROW OVERLOAD!
Cyclophosphamide induces ovarian granulosa cell ferroptosis via a mechanism associated with HO-1 and ROS-mediated mitochondrial dysfunction
Leptin-mediated downregulation of glutathione peroxidase 4 and iron overload contributing to podocyte ferroptosis in diabetic nephropathy
New Deferric Amine Compounds Efficiently Chelate Excess Iron to Treat Iron Overload Disorders and to Prevent Ferroptosis
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