PM10from Mexico City (MC) induce senescence-like phenotype and ATM, γH2A.X and p53 activation linked to oxidative stress
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2407
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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PM10from Mexico City (MC) induce senescence-like phenotype and ATM, γH2A.X and p53 activation linked to oxidative stress ; volume:7 ; number:1 ; day:5 ; month:2 ; year:2007 ; pages:1 ; date:2.2007
BMC cancer ; 7, Heft 1 (5.2.2007), 1, 2.2007
- Creator
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Sánchez-Pérez, Yesennia
- Contributor
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Gutiérrez-Ruíz, Concepción
Osornio-Vargas, Álvaro
Segura, Yazmin
Yescas, Graciela
García-Cuellar, Claudia
SpringerLink (Online service)
- DOI
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10.1186/1471-2407-7-S1-A30
- URN
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urn:nbn:de:1111-2016030345409
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:38 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sánchez-Pérez, Yesennia
- Gutiérrez-Ruíz, Concepción
- Osornio-Vargas, Álvaro
- Segura, Yazmin
- Yescas, Graciela
- García-Cuellar, Claudia
- SpringerLink (Online service)
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ΔNp63 transcriptionally regulates ATM to control p53 Serine-15 phosphorylation
FBXW7 inactivation induces cellular senescence via accumulation of p53
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Partial p53 reactivation is sufficient to induce cancer regression
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Nachweis geringer strahleninduzierter DNA-Schäden mittels automatisierter Quantifizierung von γH2AX [Gamma-H2AX]
Kernweite H2AX-Phosphorylierung nach Schwerionenbestrahlung
BTK modulates p73 activity to induce apoptosis independently of p53
Persistent repair intermediates induce senescence
ATM activation accompanies histone H2AX phosphorylation in A549 cells upon exposure to tobacco smoke
PIK3R3 inhibits cell senescence through p53/p21 signaling
ΔNp63 transcriptionally regulates ATM to control p53 Serine-15 phosphorylation
FBXW7 inactivation induces cellular senescence via accumulation of p53
Phosphorylierung von Histon H2A.X (γH2A.X) als Marker für oxidativen Stress
Partial p53 reactivation is sufficient to induce cancer regression
pATM and γH2AX are effective radiation biomarkers in assessing the radiosensitivity of 12C6+ in human tumor cells
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Nachweis geringer strahleninduzierter DNA-Schäden mittels automatisierter Quantifizierung von γH2AX [Gamma-H2AX]
Kernweite H2AX-Phosphorylierung nach Schwerionenbestrahlung
BTK modulates p73 activity to induce apoptosis independently of p53
Persistent repair intermediates induce senescence
ATM activation accompanies histone H2AX phosphorylation in A549 cells upon exposure to tobacco smoke
PIK3R3 inhibits cell senescence through p53/p21 signaling
ΔNp63 transcriptionally regulates ATM to control p53 Serine-15 phosphorylation
FBXW7 inactivation induces cellular senescence via accumulation of p53
Phosphorylierung von Histon H2A.X (γH2A.X) als Marker für oxidativen Stress
Partial p53 reactivation is sufficient to induce cancer regression
pATM and γH2AX are effective radiation biomarkers in assessing the radiosensitivity of 12C6+ in human tumor cells
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Nachweis geringer strahleninduzierter DNA-Schäden mittels automatisierter Quantifizierung von γH2AX [Gamma-H2AX]
Kernweite H2AX-Phosphorylierung nach Schwerionenbestrahlung