A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-1723
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration ; volume:12 ; number:1 ; day:10 ; month:8 ; year:2021 ; pages:1-14 ; date:12.2021
Nature Communications ; 12, Heft 1 (10.8.2021), 1-14, 12.2021
- Creator
-
Raso, Andrea
Dirkx, Ellen
Sampaio-Pinto, Vasco
Azzouzi, Hamid el
Cubero, Ryan J.
Sorensen, Daniel W.
Ottaviani, Lara
Olieslagers, Servé
Huibers, Manon M.
Weger, Roel de
Siddiqi, Sailay
Moimas, Silvia
Torrini, Consuelo
Zentillin, Lorena
Braga, Luca
Nascimento, Diana S.
Costa Martins, Paula A. da
Berlo, Jop H. van
Zacchigna, Serena
Giacca, Mauro
Windt, Leon J. De
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1038/s41467-021-25211-4
- URN
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urn:nbn:de:101:1-2022111309001395365061
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:25 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Raso, Andrea
- Dirkx, Ellen
- Sampaio-Pinto, Vasco
- Azzouzi, Hamid el
- Cubero, Ryan J.
- Sorensen, Daniel W.
- Ottaviani, Lara
- Olieslagers, Servé
- Huibers, Manon M.
- Weger, Roel de
- Siddiqi, Sailay
- Moimas, Silvia
- Torrini, Consuelo
- Zentillin, Lorena
- Braga, Luca
- Nascimento, Diana S.
- Costa Martins, Paula A. da
- Berlo, Jop H. van
- Zacchigna, Serena
- Giacca, Mauro
- Windt, Leon J. De
- SpringerLink (Online service)
Other Objects (12)
Author Correction: A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies
Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
The functional role of m6A demethylase ALKBH5 in cardiomyocyte hypertrophy
ADAP1 limits neonatal cardiomyocyte hypertrophy by reducing integrin cell surface expression
Regulation of microRNA during cardiomyocyte maturation in sheep
MRI based non-invasive detection of cardiomyocyte hypertrophy and cell-volume changes
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Author Correction: A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies
Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
The functional role of m6A demethylase ALKBH5 in cardiomyocyte hypertrophy
ADAP1 limits neonatal cardiomyocyte hypertrophy by reducing integrin cell surface expression
Regulation of microRNA during cardiomyocyte maturation in sheep
MRI based non-invasive detection of cardiomyocyte hypertrophy and cell-volume changes
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Author Correction: A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies
Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
The functional role of m6A demethylase ALKBH5 in cardiomyocyte hypertrophy
ADAP1 limits neonatal cardiomyocyte hypertrophy by reducing integrin cell surface expression
Regulation of microRNA during cardiomyocyte maturation in sheep
MRI based non-invasive detection of cardiomyocyte hypertrophy and cell-volume changes
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy