Modulating the inflammatory properties of activated microglia with Docosahexaenoic acid and Aspirin
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1476-511X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Modulating the inflammatory properties of activated microglia with Docosahexaenoic acid and Aspirin ; volume:12 ; number:1 ; day:11 ; month:2 ; year:2013 ; pages:1-14 ; date:12.2013
Lipids in health and disease ; 12, Heft 1 (11.2.2013), 1-14, 12.2013
- Creator
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Pettit, Lauren K.
- Contributor
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Varsanyi, Christopher
Tadros, James
Vassiliou, Evros
SpringerLink (Online service)
- DOI
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10.1186/1476-511X-12-16
- URN
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urn:nbn:de:1111-2016061013831
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:52 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Pettit, Lauren K.
- Varsanyi, Christopher
- Tadros, James
- Vassiliou, Evros
- SpringerLink (Online service)
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Remodeling of lipid bodies by docosahexaenoic acid in activated microglial cells
Hypoglycemia-activated Hypothalamic Microglia Impairs Glucose Counterregulatory Responses
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Docosahexaenoic acid (DHA): a modulator of microglia activity and dendritic spine morphology
β-Lapachone suppresses neuroinflammation by modulating the expression of cytokines and matrix metalloproteinases in activated microglia
Remodeling of lipid bodies by docosahexaenoic acid in activated microglial cells
Hypoglycemia-activated Hypothalamic Microglia Impairs Glucose Counterregulatory Responses
Docosahexaenoic acid-containing choline phospholipid modulates LPS-induced neuroinflammation in vivo and in microglia in vitro
Aspirin Days (Aspirin days : )
Activated microglia contribute to neuronal apoptosis in Toxoplasmic encephalitis
The role of microglia phenotypes in modulating CD4+ T cell responses
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