Sox2 overexpression alleviates noise-induced hearing loss by inhibiting inflammation-related hair cell apoptosis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-2094
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Sox2 overexpression alleviates noise-induced hearing loss by inhibiting inflammation-related hair cell apoptosis ; volume:19 ; number:1 ; day:28 ; month:2 ; year:2022 ; pages:1-16 ; date:12.2022
Journal of neuroinflammation ; 19, Heft 1 (28.2.2022), 1-16, 12.2022
- Creator
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Chen, Diyan
Jia, Gaogan
Zhang, Yanping
Mao, Huanyu
Zhao, Liping
Li, Wenyan
Chen, Yan
Ni, Yusu
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12974-022-02414-0
- URN
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urn:nbn:de:101:1-2022052820591800728075
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:33 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Chen, Diyan
- Jia, Gaogan
- Zhang, Yanping
- Mao, Huanyu
- Zhao, Liping
- Li, Wenyan
- Chen, Yan
- Ni, Yusu
- SpringerLink (Online service)
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Norepinephrine protects against cochlear outer hair cell damage and noise-induced hearing loss via α2A-adrenergic receptor
Over-expression of myosin7A in cochlear hair cells of circling mice
Noise-induced transitions in optomechanical synchronization
Ultrastructure of noise-induced cochlear synaptopathy
Generation of a biotinylatable Sox2 mouse model to identify Sox2 complexes in vivo
Human paraoxonase gene cluster overexpression alleviates angiotensin II-induced cardiac hypertrophy in mice
FOXP2 overexpression upregulates LAMA4 expression and thereby alleviates preeclampsia by regulating trophoblast behavior
Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss
Self-restricted circular RNA circSOX2 suppressed the malignant progression in SOX2-amplified LUSC
AAV-mediated NT-3 overexpression protects cochleae against noise-induced synaptopathy
Activated tissue-resident macrophages contribute to hair cell insults in noise-induced hearing loss in mice
Overexpression of apolipoprotein A-I alleviates endoplasmic reticulum stress in hepatocytes
Norepinephrine protects against cochlear outer hair cell damage and noise-induced hearing loss via α2A-adrenergic receptor
Over-expression of myosin7A in cochlear hair cells of circling mice
Noise-induced transitions in optomechanical synchronization
Ultrastructure of noise-induced cochlear synaptopathy
Generation of a biotinylatable Sox2 mouse model to identify Sox2 complexes in vivo
Human paraoxonase gene cluster overexpression alleviates angiotensin II-induced cardiac hypertrophy in mice
FOXP2 overexpression upregulates LAMA4 expression and thereby alleviates preeclampsia by regulating trophoblast behavior
Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss
Self-restricted circular RNA circSOX2 suppressed the malignant progression in SOX2-amplified LUSC
AAV-mediated NT-3 overexpression protects cochleae against noise-induced synaptopathy
Activated tissue-resident macrophages contribute to hair cell insults in noise-induced hearing loss in mice
Overexpression of apolipoprotein A-I alleviates endoplasmic reticulum stress in hepatocytes
Norepinephrine protects against cochlear outer hair cell damage and noise-induced hearing loss via α2A-adrenergic receptor
Over-expression of myosin7A in cochlear hair cells of circling mice
Noise-induced transitions in optomechanical synchronization
Ultrastructure of noise-induced cochlear synaptopathy
Generation of a biotinylatable Sox2 mouse model to identify Sox2 complexes in vivo
Human paraoxonase gene cluster overexpression alleviates angiotensin II-induced cardiac hypertrophy in mice
FOXP2 overexpression upregulates LAMA4 expression and thereby alleviates preeclampsia by regulating trophoblast behavior
Overexpression of the receptor for advanced glycation end-products in the auditory cortex of rats with noise-induced hearing loss