ubtor Mutation Causes Motor Hyperactivity by Activating mTOR Signaling in Zebrafish

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1995-8218

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: ubtor Mutation Causes Motor Hyperactivity by Activating mTOR Signaling in Zebrafish ; day:26 ; month:7 ; year:2021 ; pages:1-13
Neuroscience bulletin ; (26.7.2021), 1-13

Creator: Wang, Tiantian
Zhou, Mingshan
Zhang, Quan
Zhang, Cuizhen
Peng, Gang

Contributor: SpringerLink (Online service)

DOI: 10.1007/s12264-021-00755-z

URN: urn:nbn:de:101:1-2021102408540360761670

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:35 AM CEST

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Wang, Tiantian
Zhou, Mingshan
Zhang, Quan
Zhang, Cuizhen
Peng, Gang
SpringerLink (Online service)

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CSNK2B contributes to colorectal cancer cell proliferation by activating the mTOR signaling

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Zebrafish heart regeneration: the roles of DNA damage response and mTor signaling

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Asxl1 C-terminal mutation perturbs neutrophil differentiation in zebrafish

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ubtor Mutation Causes Motor Hyperactivity by Activating mTOR Signaling in Zebrafish

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Other Objects (12)

mTOR mutation disrupts larval zebrafish tail fin regeneration via regulating proliferation of blastema cells and mitochondrial functions

CSNK2B contributes to colorectal cancer cell proliferation by activating the mTOR signaling

Zebrafish heart regeneration: the roles of DNA damage response and mTor signaling

USP5 knockdown alleviates lung cancer progression via activating PARP1-mediated mTOR signaling pathway

Golgi phosphoprotein 3 (GOLPH3) promotes hepatocellular carcinoma progression by activating mTOR signaling pathway

Multifocal, multiphenotypic tumours arising from an MTOR mutation acquired in early embryogenesis

Activating mutation in MET oncogene in familial colorectal cancer

Loss of pex5 sensitizes zebrafish to fasting due to deregulated mitochondria, mTOR, and autophagy

Asxl1 C-terminal mutation perturbs neutrophil differentiation in zebrafish

The role of metabolic ecosystem in cancer progression — metabolic plasticity and mTOR hyperactivity in tumor tissues

Recurrent activating STAT5B N642H mutation in myeloid neoplasms with eosinophilia

Effects of nicotinic acetylcholine receptor-activating alkaloids on anxiety-like behavior in zebrafish

mTOR mutation disrupts larval zebrafish tail fin regeneration via regulating proliferation of blastema cells and mitochondrial functions

CSNK2B contributes to colorectal cancer cell proliferation by activating the mTOR signaling

Zebrafish heart regeneration: the roles of DNA damage response and mTor signaling

USP5 knockdown alleviates lung cancer progression via activating PARP1-mediated mTOR signaling pathway

Golgi phosphoprotein 3 (GOLPH3) promotes hepatocellular carcinoma progression by activating mTOR signaling pathway

Multifocal, multiphenotypic tumours arising from an MTOR mutation acquired in early embryogenesis

Activating mutation in MET oncogene in familial colorectal cancer

Loss of pex5 sensitizes zebrafish to fasting due to deregulated mitochondria, mTOR, and autophagy

Asxl1 C-terminal mutation perturbs neutrophil differentiation in zebrafish

The role of metabolic ecosystem in cancer progression — metabolic plasticity and mTOR hyperactivity in tumor tissues

Recurrent activating STAT5B N642H mutation in myeloid neoplasms with eosinophilia

Effects of nicotinic acetylcholine receptor-activating alkaloids on anxiety-like behavior in zebrafish

mTOR mutation disrupts larval zebrafish tail fin regeneration via regulating proliferation of blastema cells and mitochondrial functions

CSNK2B contributes to colorectal cancer cell proliferation by activating the mTOR signaling

Zebrafish heart regeneration: the roles of DNA damage response and mTor signaling

USP5 knockdown alleviates lung cancer progression via activating PARP1-mediated mTOR signaling pathway

Golgi phosphoprotein 3 (GOLPH3) promotes hepatocellular carcinoma progression by activating mTOR signaling pathway

Multifocal, multiphenotypic tumours arising from an MTOR mutation acquired in early embryogenesis

Activating mutation in MET oncogene in familial colorectal cancer

Loss of pex5 sensitizes zebrafish to fasting due to deregulated mitochondria, mTOR, and autophagy

Asxl1 C-terminal mutation perturbs neutrophil differentiation in zebrafish

The role of metabolic ecosystem in cancer progression — metabolic plasticity and mTOR hyperactivity in tumor tissues

Recurrent activating STAT5B N642H mutation in myeloid neoplasms with eosinophilia

Effects of nicotinic acetylcholine receptor-activating alkaloids on anxiety-like behavior in zebrafish

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