Chromosome 1q gain and tenascin-C expression are candidate markers to define different risk groups in pediatric posterior fossa ependymoma
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2051-5960
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Chromosome 1q gain and tenascin-C expression are candidate markers to define different risk groups in pediatric posterior fossa ependymoma ; volume:4 ; number:1 ; day:22 ; month:8 ; year:2016 ; pages:1-10 ; date:12.2016
Acta Neuropathologica Communications ; 4, Heft 1 (22.8.2016), 1-10, 12.2016
- Creator
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Araki, Asuka
- Contributor
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Chocholous, Monika
Gojo, Johannes
Dorfer, Christian
Czech, Thomas
Heinzl, Harald
Dieckmann, Karin
Ambros, Inge M.
Ambros, Peter F.
Slavc, Irene
Haberler, Christine
SpringerLink (Online service)
- DOI
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10.1186/s40478-016-0349-9
- URN
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urn:nbn:de:1111-201609209425
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:55 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Araki, Asuka
- Chocholous, Monika
- Gojo, Johannes
- Dorfer, Christian
- Czech, Thomas
- Heinzl, Harald
- Dieckmann, Karin
- Ambros, Inge M.
- Ambros, Peter F.
- Slavc, Irene
- Haberler, Christine
- SpringerLink (Online service)
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EZHIP: a new piece of the puzzle towards understanding pediatric posterior fossa ependymoma
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Improved risk-stratification for posterior fossa ependymoma of childhood considering clinical, histological and genetic features – a retrospective analysis of the HIT ependymoma trial cohort
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NF2 and ZFTA evaluation in the diagnostic algorithm of pediatric posterior fossa ependymoma with H3K27ME3 retained expression
Identification of key methylation differentially expressed genes in posterior fossa ependymoma based on epigenomic and transcriptome analysis
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