Highly activated p53 contributes to selectively increased apoptosis of latently HIV-1 infected cells upon treatment of anticancer drugs
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1743-422X
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Highly activated p53 contributes to selectively increased apoptosis of latently HIV-1 infected cells upon treatment of anticancer drugs ; volume:13 ; number:1 ; day:16 ; month:8 ; year:2016 ; pages:1-9 ; date:12.2016
Virology journal ; 13, Heft 1 (16.8.2016), 1-9, 12.2016
- Urheber
-
Shin, YoungHyun
- Beteiligte Personen und Organisationen
-
Lim, Hoyong
Choi, Byeong-Sun
Kim, Kyung-Chang
Kang, Chun
Bae, Yong-Soo
Yoon, Cheol-Hee
SpringerLink (Online service)
- DOI
-
10.1186/s12985-016-0595-2
- URN
-
urn:nbn:de:1111-20160913504
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:54 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Shin, YoungHyun
- Lim, Hoyong
- Choi, Byeong-Sun
- Kim, Kyung-Chang
- Kang, Chun
- Bae, Yong-Soo
- Yoon, Cheol-Hee
- SpringerLink (Online service)
Ähnliche Objekte (12)
Explicit contextual information selectively contributes to predictive switching of internal models
Generation of a selectively cytotoxic fusion protein against p53 mutated cancers
Ruthenium and Gold Complexes as potential Anticancer Drugs targeting selectively Integrin Receptors
Targeting the p53-MDM2 and p53-MDMX interaction as a therapeutic strategy for anticancer treatment
Targeting the p53-MDM2 and p53-MDMX interaction as a therapeutic strategy for anticancer treatment
Kayadiol exerted anticancer effects through p53-mediated ferroptosis in NKTCL cells
Genomic instability in mutant p53 cancer cells upon entotic engulfment
Genetic inhibition of the atypical kinase Wee1 selectively drives apoptosis of p53 inactive tumor cells
Aminobenzothiazole derivatives stabilize the thermolabile p53 cancer mutant Y220C and show anticancer activity in p53-Y220C cell lines
P53/microRNA-34-induced metabolic regulation: new opportunities in anticancer therapy
BMAL1/p53 mediating bronchial epithelial cell autophagy contributes to PM2.5-aggravated asthma
Regulation of GAD65 expression by SMAR1 and p53 upon Streptozotocin treatment
Explicit contextual information selectively contributes to predictive switching of internal models
Generation of a selectively cytotoxic fusion protein against p53 mutated cancers
Ruthenium and Gold Complexes as potential Anticancer Drugs targeting selectively Integrin Receptors
Targeting the p53-MDM2 and p53-MDMX interaction as a therapeutic strategy for anticancer treatment
Targeting the p53-MDM2 and p53-MDMX interaction as a therapeutic strategy for anticancer treatment
Kayadiol exerted anticancer effects through p53-mediated ferroptosis in NKTCL cells
Genomic instability in mutant p53 cancer cells upon entotic engulfment
Genetic inhibition of the atypical kinase Wee1 selectively drives apoptosis of p53 inactive tumor cells
Aminobenzothiazole derivatives stabilize the thermolabile p53 cancer mutant Y220C and show anticancer activity in p53-Y220C cell lines
P53/microRNA-34-induced metabolic regulation: new opportunities in anticancer therapy
BMAL1/p53 mediating bronchial epithelial cell autophagy contributes to PM2.5-aggravated asthma
Regulation of GAD65 expression by SMAR1 and p53 upon Streptozotocin treatment
Explicit contextual information selectively contributes to predictive switching of internal models
Generation of a selectively cytotoxic fusion protein against p53 mutated cancers
Ruthenium and Gold Complexes as potential Anticancer Drugs targeting selectively Integrin Receptors
Targeting the p53-MDM2 and p53-MDMX interaction as a therapeutic strategy for anticancer treatment
Targeting the p53-MDM2 and p53-MDMX interaction as a therapeutic strategy for anticancer treatment
Kayadiol exerted anticancer effects through p53-mediated ferroptosis in NKTCL cells
Genomic instability in mutant p53 cancer cells upon entotic engulfment
Genetic inhibition of the atypical kinase Wee1 selectively drives apoptosis of p53 inactive tumor cells
Aminobenzothiazole derivatives stabilize the thermolabile p53 cancer mutant Y220C and show anticancer activity in p53-Y220C cell lines
P53/microRNA-34-induced metabolic regulation: new opportunities in anticancer therapy
BMAL1/p53 mediating bronchial epithelial cell autophagy contributes to PM2.5-aggravated asthma