Erratum to: triple-negative, basal-like, and quintuple-negative breast cancers: better prediction model for survival

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2407

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Erratum to: triple-negative, basal-like, and quintuple-negative breast cancers: better prediction model for survival ; volume:11 ; number:1 ; day:12 ; month:1 ; year:2011 ; pages:1-2 ; date:12.2011
BMC cancer ; 11, Heft 1 (12.1.2011), 1-2, 12.2011

Creator: Choi, Yoon-La

Contributor: Oh, Ensel
Park, Sarah
Kim, Yeonju
Park, Yeon-Hee
Song, Kyug
Cho, Eun Yoon
Hong, Yun-Chul
Choi, Jong Sun
Lee, Jeong Eon
Kim, Jung Han
Nam, Seok Jin
Lim, Young-Hyuck
Yang, Jung-Hyun
Shin, Young Kee
SpringerLink (Online service)

DOI: 10.1186/1471-2407-11-13

URN: urn:nbn:de:1111-2016030365007

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:25 AM CEST

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Associated

Choi, Yoon-La
Oh, Ensel
Park, Sarah
Kim, Yeonju
Park, Yeon-Hee
Song, Kyug
Cho, Eun Yoon
Hong, Yun-Chul
Choi, Jong Sun
Lee, Jeong Eon
Kim, Jung Han
Nam, Seok Jin
Lim, Young-Hyuck
Yang, Jung-Hyun
Shin, Young Kee
SpringerLink (Online service)

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Erratum to: triple-negative, basal-like, and quintuple-negative breast cancers: better prediction model for survival

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Other Objects (12)

Triple-negative, basal-like, and quintuple-negative breast cancers: better prediction model for survival

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Knockdown of CAVEOLIN-1 Sensitizes Human Basal-Like Triple-Negative Breast Cancer Cells to Radiation

Oxidative stress and counteracting mechanisms in hormone receptor positive, triple-negative and basal-like breast carcinomas

Basal-like phenotype is not associated with patient survival in estrogen-receptor-negative breast cancers

SHP2 acts both upstream and downstream of multiple receptor tyrosine kinases to promote basal-like and triple-negative breast cancer

Inhibition of SHP2 in basal-like and triple-negative breast cells induces basal-to-luminal transition, hormone dependency, and sensitivity to anti-hormone treatment

A clinically relevant gene signature in triple negative and basal-like breast cancer

A clinically relevant gene signature in triple negative and basal-like breast cancer

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Targeting a cell state common to triple‐negative breast cancers

Comparative microRNA profiling of sporadic and BRCA1 associated basal-like breast cancers

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Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Knockdown of CAVEOLIN-1 Sensitizes Human Basal-Like Triple-Negative Breast Cancer Cells to Radiation

Oxidative stress and counteracting mechanisms in hormone receptor positive, triple-negative and basal-like breast carcinomas

Basal-like phenotype is not associated with patient survival in estrogen-receptor-negative breast cancers

SHP2 acts both upstream and downstream of multiple receptor tyrosine kinases to promote basal-like and triple-negative breast cancer

Inhibition of SHP2 in basal-like and triple-negative breast cells induces basal-to-luminal transition, hormone dependency, and sensitivity to anti-hormone treatment

A clinically relevant gene signature in triple negative and basal-like breast cancer

A clinically relevant gene signature in triple negative and basal-like breast cancer

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Targeting a cell state common to triple‐negative breast cancers

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Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Knockdown of CAVEOLIN-1 Sensitizes Human Basal-Like Triple-Negative Breast Cancer Cells to Radiation

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