Hochschulschrift
Hyperforin induces senescence in endothelial and tumor cells
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Maße
-
30 cm
- Umfang
-
83 Seiten
- Sprache
-
Englisch
- Anmerkungen
-
Illustrationen, Diagramme
Ruperto-Carola University of Heidelberg, Dissertation, 2015
- Klassifikation
-
Biowissenschaften, Biologie
- Inhaltsverzeichnis
- Rechteinformation
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Letzte Aktualisierung
-
11.06.2025, 13:35 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Objekttyp
- Hochschulschrift
Beteiligte
Entstanden
- [2015]
Ähnliche Objekte (12)
Hyperforin induces senescence in endothelial and tumor cells
Prelamin accumulation in primary endothelial cells induces premature senescence and activation
The role of endothelial senescence in malignant tumors
Innate immune cell crosstalk induces melanoma cell senescence
FK506 induces lung lymphatic endothelial cell senescence and downregulates LYVE-1 expression, with associated decreased hyaluronan uptake
Endothelial senescence and microRNA
Oxidative glucose metabolism promotes senescence in vascular endothelial cells
The role of the proteasome in endothelial cell senescence
Isolation of circulating endothelial cells provides tool to determine endothelial cell senescence in blood samples
Hepatocellular senescence induces multi-organ senescence and dysfunction via TGFβ
Telomeric DNA induces apoptosis and senescence of human breast carcinoma cells
MEF2A alters the proliferation, inflammation-related gene expression profiles and its silencing induces cellular senescence in human coronary endothelial cells
Hyperforin induces senescence in endothelial and tumor cells
Prelamin accumulation in primary endothelial cells induces premature senescence and activation
The role of endothelial senescence in malignant tumors
Innate immune cell crosstalk induces melanoma cell senescence
FK506 induces lung lymphatic endothelial cell senescence and downregulates LYVE-1 expression, with associated decreased hyaluronan uptake
Endothelial senescence and microRNA
Oxidative glucose metabolism promotes senescence in vascular endothelial cells
The role of the proteasome in endothelial cell senescence
Isolation of circulating endothelial cells provides tool to determine endothelial cell senescence in blood samples
Hepatocellular senescence induces multi-organ senescence and dysfunction via TGFβ
Telomeric DNA induces apoptosis and senescence of human breast carcinoma cells
MEF2A alters the proliferation, inflammation-related gene expression profiles and its silencing induces cellular senescence in human coronary endothelial cells
Hyperforin induces senescence in endothelial and tumor cells
Prelamin accumulation in primary endothelial cells induces premature senescence and activation
The role of endothelial senescence in malignant tumors
Innate immune cell crosstalk induces melanoma cell senescence
FK506 induces lung lymphatic endothelial cell senescence and downregulates LYVE-1 expression, with associated decreased hyaluronan uptake
Endothelial senescence and microRNA
Oxidative glucose metabolism promotes senescence in vascular endothelial cells
The role of the proteasome in endothelial cell senescence
Isolation of circulating endothelial cells provides tool to determine endothelial cell senescence in blood samples
Hepatocellular senescence induces multi-organ senescence and dysfunction via TGFβ
Telomeric DNA induces apoptosis and senescence of human breast carcinoma cells