Removal of H2A.Z by INO 80 promotes homologous recombination

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: Removal of H2A.Z by INO 80 promotes homologous recombination ; volume:16 ; number:8 ; year:2015 ; pages:986-994 ; extent:9
EMBO reports / European Molecular Biology Organization ; 16, Heft 8 (2015), 986-994 (gesamt 9)

Creator: Alatwi, Hanan E.
Downs, Jessica A.

DOI: 10.15252/embr.201540330

URN: urn:nbn:de:101:1-2022120204343852158646

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:25 AM CEST

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Alatwi, Hanan E.
Downs, Jessica A.

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Structural basis of homologous recombination

RAD18 O-GlcNAcylation promotes translesion DNA synthesis and homologous recombination repair

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Leveraging homologous recombination repair deficiency in sarcoma

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Pan-cancer landscape of homologous recombination deficiency

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ZGRF1 promotes end resection of DNA homologous recombination via forming complex with BRCA1/EXO1

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Structural basis of homologous recombination

RAD18 O-GlcNAcylation promotes translesion DNA synthesis and homologous recombination repair

Homologous recombination deficiency in breast cancer

Leveraging homologous recombination repair deficiency in sarcoma

Targeting homologous recombination deficiency in uterine leiomyosarcoma

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Opposing roles for 53BP1 during homologous recombination

Pan-cancer landscape of homologous recombination deficiency

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KNO1‐mediated autophagic degradation of the Bloom syndrome complex component RMI1 promotes homologous recombination

ZGRF1 promotes end resection of DNA homologous recombination via forming complex with BRCA1/EXO1

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Removal of H2A.Z by INO 80 promotes homologous recombination

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Other Objects (12)

Removal of TREX1 activity enhances CRISPR–Cas9-mediated homologous recombination

Structural basis of homologous recombination

RAD18 O-GlcNAcylation promotes translesion DNA synthesis and homologous recombination repair

Homologous recombination deficiency in breast cancer

Leveraging homologous recombination repair deficiency in sarcoma

Targeting homologous recombination deficiency in uterine leiomyosarcoma

Homologous recombination deficiency in epithelial ovarian cancer

Opposing roles for 53BP1 during homologous recombination

Pan-cancer landscape of homologous recombination deficiency

Homologous Recombination Subpathways: A Tangle to Resolve

KNO1‐mediated autophagic degradation of the Bloom syndrome complex component RMI1 promotes homologous recombination

ZGRF1 promotes end resection of DNA homologous recombination via forming complex with BRCA1/EXO1

Removal of TREX1 activity enhances CRISPR–Cas9-mediated homologous recombination

Structural basis of homologous recombination

RAD18 O-GlcNAcylation promotes translesion DNA synthesis and homologous recombination repair

Homologous recombination deficiency in breast cancer

Leveraging homologous recombination repair deficiency in sarcoma

Targeting homologous recombination deficiency in uterine leiomyosarcoma

Homologous recombination deficiency in epithelial ovarian cancer

Opposing roles for 53BP1 during homologous recombination

Pan-cancer landscape of homologous recombination deficiency

Homologous Recombination Subpathways: A Tangle to Resolve

KNO1‐mediated autophagic degradation of the Bloom syndrome complex component RMI1 promotes homologous recombination

ZGRF1 promotes end resection of DNA homologous recombination via forming complex with BRCA1/EXO1

Removal of TREX1 activity enhances CRISPR–Cas9-mediated homologous recombination

Structural basis of homologous recombination

RAD18 O-GlcNAcylation promotes translesion DNA synthesis and homologous recombination repair

Homologous recombination deficiency in breast cancer

Leveraging homologous recombination repair deficiency in sarcoma

Targeting homologous recombination deficiency in uterine leiomyosarcoma

Homologous recombination deficiency in epithelial ovarian cancer

Opposing roles for 53BP1 during homologous recombination

Pan-cancer landscape of homologous recombination deficiency

Homologous Recombination Subpathways: A Tangle to Resolve

KNO1‐mediated autophagic degradation of the Bloom syndrome complex component RMI1 promotes homologous recombination

ZGRF1 promotes end resection of DNA homologous recombination via forming complex with BRCA1/EXO1

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