- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
In: iScience. Elsevier. 2024, 27(9), 110814. eISSN 2589-0042. Verfügbar unter: doi: 10.1016/j.isci.2024.110814
- Event
-
Veröffentlichung
- (where)
-
Konstanz
- (who)
-
KOPS Universität Konstanz
- (when)
-
2024
- Creator
- URN
-
urn:nbn:de:bsz:352-2-sgei13y0tl6k5
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:31 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Tanigawa, Mirai
- Maeda, Tatsuya
- Isono, Erika
- KOPS Universität Konstanz
Time of origin
- 2024
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The nutrient-responsive CDK Pho85 primes the Sch9 kinase for its activation by TORC1
Analysis of VEGF-responsive Genes Involved in the activation of endothelial cells
ArfGAP1 inhibits mTORC1 lysosomal localization and activation
Ubiquitination of Rheb governs growth factor-induced mTORC1 activation
High glutamine suppresses osteogenesis through mTORC1-mediated inhibition of the mTORC2/AKT-473/RUNX2 axis
USP32-regulated LAMTOR1 ubiquitination impacts mTORC1 activation and autophagy induction
ATP6V0d2 mediates leucine-induced mTORC1 activation and polarization of macrophages
USP32-regulated LAMTOR1 ubiquitination impacts mTORC1 activation and autophagy induction
The sensitivity of mTORC1 signaling activation renders tissue regenerative capacity
Co-ingestion of glutamine and leucine synergistically promotes mTORC1 activation
Inhibition of glutamine uptake regulates mTORC1, glutamine metabolism and cell growth in prostate cancer
Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration
The nutrient-responsive CDK Pho85 primes the Sch9 kinase for its activation by TORC1
Analysis of VEGF-responsive Genes Involved in the activation of endothelial cells
ArfGAP1 inhibits mTORC1 lysosomal localization and activation
Ubiquitination of Rheb governs growth factor-induced mTORC1 activation
High glutamine suppresses osteogenesis through mTORC1-mediated inhibition of the mTORC2/AKT-473/RUNX2 axis
USP32-regulated LAMTOR1 ubiquitination impacts mTORC1 activation and autophagy induction
ATP6V0d2 mediates leucine-induced mTORC1 activation and polarization of macrophages
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