Overweight worsens apoptosis, neuroinflammation and blood-brain barrier damage after hypoxic ischemia in neonatal brain through JNK hyperactivation
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1742-2094
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Overweight worsens apoptosis, neuroinflammation and blood-brain barrier damage after hypoxic ischemia in neonatal brain through JNK hyperactivation ; volume:8 ; number:1 ; day:25 ; month:4 ; year:2011 ; pages:1-15 ; date:12.2011
Journal of neuroinflammation ; 8, Heft 1 (25.4.2011), 1-15, 12.2011
- Urheber
-
Tu, Yi-Fang
- Beteiligte Personen und Organisationen
-
Tsai, Yau-Sheng
Wang, Lan-Wan
Wu, Hsin-Chieh
Huang, Chao-Ching
Ho, Chien-Jung
SpringerLink (Online service)
- DOI
-
10.1186/1742-2094-8-40
- URN
-
urn:nbn:de:1111-2016032329777
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:55 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Tu, Yi-Fang
- Tsai, Yau-Sheng
- Wang, Lan-Wan
- Wu, Hsin-Chieh
- Huang, Chao-Ching
- Ho, Chien-Jung
- SpringerLink (Online service)
Ähnliche Objekte (12)
Neuro-inflammation, blood-brain barrier, seizures and autism
JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain
Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment
TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain
Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice
Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis
Brain endothelial LRP1 maintains blood–brain barrier integrity
Neuroinflammation and Brain Disease
NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice
ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury
Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis
Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation
Neuro-inflammation, blood-brain barrier, seizures and autism
JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain
Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment
TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain
Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice
Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis
Brain endothelial LRP1 maintains blood–brain barrier integrity
Neuroinflammation and Brain Disease
NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice
ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury
Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis
Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation
Neuro-inflammation, blood-brain barrier, seizures and autism
JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain
Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment
TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain
Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice
Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis
Brain endothelial LRP1 maintains blood–brain barrier integrity
Neuroinflammation and Brain Disease
NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice
ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury
Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis