Suppression of interferon gene expression overcomes resistance to MEK inhibition in KRAS-mutant colorectal cancer
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1476-5594
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Suppression of interferon gene expression overcomes resistance to MEK inhibition in KRAS-mutant colorectal cancer ; volume:38 ; number:10 ; day:23 ; month:10 ; year:2018 ; pages:1717-1733 ; date:3.2019
Oncogene ; 38, Heft 10 (23.10.2018), 1717-1733, 3.2019
- Creator
-
Wagner, Steve
Vlachogiannis, Georgios
Haven Brandon, Alexis De
Valenti, Melanie
Box, Gary
Jenkins, Liam
Mancusi, Caterina
Self, Annette
Manodoro, Floriana
Assiotis, Ioannis
Robinson, Penny
Chauhan, Ritika
Rust, Alistair G.
Matthews, Nik
Eason, Kate
Khan, Khurum
Starling, Naureen
Cunningham, David
Sadanandam, Anguraj
Isacke, Clare M.
Kirkin, Vladimir
Valeri, Nicola
Whittaker, Steven R.
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1038/s41388-018-0554-z
- URN
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urn:nbn:de:101:1-2019110502335033783680
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:19 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wagner, Steve
- Vlachogiannis, Georgios
- Haven Brandon, Alexis De
- Valenti, Melanie
- Box, Gary
- Jenkins, Liam
- Mancusi, Caterina
- Self, Annette
- Manodoro, Floriana
- Assiotis, Ioannis
- Robinson, Penny
- Chauhan, Ritika
- Rust, Alistair G.
- Matthews, Nik
- Eason, Kate
- Khan, Khurum
- Starling, Naureen
- Cunningham, David
- Sadanandam, Anguraj
- Isacke, Clare M.
- Kirkin, Vladimir
- Valeri, Nicola
- Whittaker, Steven R.
- SpringerLink (Online service)
Other Objects (12)
MEK nuclear localization promotes YAP stability via sequestering β-TrCP in KRAS mutant cancer cells
Dual inhibition of SUMOylation and MEK conquers MYC-expressing KRAS-mutant cancers by accumulating DNA damage
Small Molecule KRAS Agonist for Mutant KRAS Cancer Therapy
Comprehensive antitumor immune response boosted by dual inhibition of SUMOylation and MEK in MYC-expressing KRAS-mutant cancers
Target Mutant KRAS with Molecular Glue
A combination therapy for KRAS-mutant lung cancer by targeting synthetic lethal partners of mutant KRAS
Correction to: Small Molecule KRAS Agonist for Mutant KRAS Cancer Therapy
A pan-KRAS degrader for the treatment of KRAS-mutant cancers
Current therapy of KRAS-mutant lung cancer
RAS-ON inhibition overcomes clinical resistance to KRAS G12C-OFF covalent blockade
Restoration of mutant K-Ras repressed miR-199b inhibits K-Ras mutant non-small cell lung cancer progression
Charakterisierung der Phosphatase PTPRO als MEK-Inhibitor-Primärresistenzmechanismus im KRAS-mutierten Kolonkarzinom
MEK nuclear localization promotes YAP stability via sequestering β-TrCP in KRAS mutant cancer cells
Dual inhibition of SUMOylation and MEK conquers MYC-expressing KRAS-mutant cancers by accumulating DNA damage
Small Molecule KRAS Agonist for Mutant KRAS Cancer Therapy
Comprehensive antitumor immune response boosted by dual inhibition of SUMOylation and MEK in MYC-expressing KRAS-mutant cancers
Target Mutant KRAS with Molecular Glue
A combination therapy for KRAS-mutant lung cancer by targeting synthetic lethal partners of mutant KRAS
Correction to: Small Molecule KRAS Agonist for Mutant KRAS Cancer Therapy
A pan-KRAS degrader for the treatment of KRAS-mutant cancers
Current therapy of KRAS-mutant lung cancer
RAS-ON inhibition overcomes clinical resistance to KRAS G12C-OFF covalent blockade
Restoration of mutant K-Ras repressed miR-199b inhibits K-Ras mutant non-small cell lung cancer progression
Charakterisierung der Phosphatase PTPRO als MEK-Inhibitor-Primärresistenzmechanismus im KRAS-mutierten Kolonkarzinom
MEK nuclear localization promotes YAP stability via sequestering β-TrCP in KRAS mutant cancer cells
Dual inhibition of SUMOylation and MEK conquers MYC-expressing KRAS-mutant cancers by accumulating DNA damage
Small Molecule KRAS Agonist for Mutant KRAS Cancer Therapy
Comprehensive antitumor immune response boosted by dual inhibition of SUMOylation and MEK in MYC-expressing KRAS-mutant cancers
Target Mutant KRAS with Molecular Glue
A combination therapy for KRAS-mutant lung cancer by targeting synthetic lethal partners of mutant KRAS
Correction to: Small Molecule KRAS Agonist for Mutant KRAS Cancer Therapy
A pan-KRAS degrader for the treatment of KRAS-mutant cancers
Current therapy of KRAS-mutant lung cancer
RAS-ON inhibition overcomes clinical resistance to KRAS G12C-OFF covalent blockade
Restoration of mutant K-Ras repressed miR-199b inhibits K-Ras mutant non-small cell lung cancer progression