The anti-estrogen receptor drug, tamoxifen, is selectively Lethal to P-glycoprotein-expressing Multidrug resistant tumor cells
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2407
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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The anti-estrogen receptor drug, tamoxifen, is selectively Lethal to P-glycoprotein-expressing Multidrug resistant tumor cells ; volume:23 ; number:1 ; day:6 ; month:1 ; year:2023 ; pages:1-14 ; date:12.2023
BMC cancer ; 23, Heft 1 (6.1.2023), 1-14, 12.2023
- Creator
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Bakadlag, Rowa
Limniatis, Georgia
Georges, Gabriel
Georges, Elias
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12885-022-10474-x
- URN
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urn:nbn:de:101:1-2023031521360554016096
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:44 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Bakadlag, Rowa
- Limniatis, Georgia
- Georges, Gabriel
- Georges, Elias
- SpringerLink (Online service)
Other Objects (12)
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Raptor localization predicts prognosis and tamoxifen response in estrogen receptor-positive breast cancer
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Predicting prognosis using molecular profiling in estrogen receptor-positive breast cancer treated with tamoxifen
Author Correction: GATA3 and MDM2 are synthetic lethal in estrogen receptor-positive breast cancers
An attachment glycoprotein nanoparticle elicits broadly neutralizing antibodies and protects against lethal Nipah virus infection
Nogo-B receptor increases the resistance to tamoxifen in estrogen receptor-positive breast cancer cells
Synthesis of Tamoxifen‐Artemisinin and Estrogen‐Artemisinin Hybrids Highly Potent Against Breast and Prostate Cancer
The Effect of Selective Estrogen Receptor Modulators (SERMs) on the Tamoxifen Resistant Breast Cancer Cells
A model of estrogen-related gene expression reveals non-linear effects in transcriptional response to tamoxifen
Targeting c-Jun inhibits fatty acid oxidation to overcome tamoxifen resistance in estrogen receptor-positive breast cancer
Tamoxifen stimulates arachidonic acid release from rat liver cells by an estrogen receptor-independent, non-genomic mechanism
GATA3 and MDM2 are synthetic lethal in estrogen receptor-positive breast cancers
Raptor localization predicts prognosis and tamoxifen response in estrogen receptor-positive breast cancer
Tamoxifen
Predicting prognosis using molecular profiling in estrogen receptor-positive breast cancer treated with tamoxifen
Author Correction: GATA3 and MDM2 are synthetic lethal in estrogen receptor-positive breast cancers
An attachment glycoprotein nanoparticle elicits broadly neutralizing antibodies and protects against lethal Nipah virus infection
Nogo-B receptor increases the resistance to tamoxifen in estrogen receptor-positive breast cancer cells
Synthesis of Tamoxifen‐Artemisinin and Estrogen‐Artemisinin Hybrids Highly Potent Against Breast and Prostate Cancer
The Effect of Selective Estrogen Receptor Modulators (SERMs) on the Tamoxifen Resistant Breast Cancer Cells
A model of estrogen-related gene expression reveals non-linear effects in transcriptional response to tamoxifen
Targeting c-Jun inhibits fatty acid oxidation to overcome tamoxifen resistance in estrogen receptor-positive breast cancer
Tamoxifen stimulates arachidonic acid release from rat liver cells by an estrogen receptor-independent, non-genomic mechanism
GATA3 and MDM2 are synthetic lethal in estrogen receptor-positive breast cancers
Raptor localization predicts prognosis and tamoxifen response in estrogen receptor-positive breast cancer
Tamoxifen
Predicting prognosis using molecular profiling in estrogen receptor-positive breast cancer treated with tamoxifen
Author Correction: GATA3 and MDM2 are synthetic lethal in estrogen receptor-positive breast cancers
An attachment glycoprotein nanoparticle elicits broadly neutralizing antibodies and protects against lethal Nipah virus infection
Nogo-B receptor increases the resistance to tamoxifen in estrogen receptor-positive breast cancer cells
Synthesis of Tamoxifen‐Artemisinin and Estrogen‐Artemisinin Hybrids Highly Potent Against Breast and Prostate Cancer
The Effect of Selective Estrogen Receptor Modulators (SERMs) on the Tamoxifen Resistant Breast Cancer Cells
A model of estrogen-related gene expression reveals non-linear effects in transcriptional response to tamoxifen
Targeting c-Jun inhibits fatty acid oxidation to overcome tamoxifen resistance in estrogen receptor-positive breast cancer