Replication of epistatic DNA loci in two case-control GWAS studies using OPE algorithm
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1471-2105
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Replication of epistatic DNA loci in two case-control GWAS studies using OPE algorithm ; volume:12 ; number:11 ; day:21 ; month:11 ; year:2011 ; pages:1-2 ; date:12.2011
BMC bioinformatics ; 12, Heft 11 (21.11.2011), 1-2, 12.2011
- Urheber
-
Goudey, Benjamin
- Beteiligte Personen und Organisationen
-
Wang, Qiao
Rawlinson, Dave
Zarnegar, Armita
Kikianty, Eder
Markham, John
Macintyre, Geoff
Abraham, Gad
Stern, Linda
Inouye, Michael
Haviv, Izhak
Kowalczyk, Adam
SpringerLink (Online service)
- DOI
-
10.1186/1471-2105-12-S11-A5
- URN
-
urn:nbn:de:1111-2016021730994
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:27 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Goudey, Benjamin
- Wang, Qiao
- Rawlinson, Dave
- Zarnegar, Armita
- Kikianty, Eder
- Markham, John
- Macintyre, Geoff
- Abraham, Gad
- Stern, Linda
- Inouye, Michael
- Haviv, Izhak
- Kowalczyk, Adam
- SpringerLink (Online service)
Ähnliche Objekte (12)
GWIS - model-free, fast and exhaustive search for epistatic interactions in case-control GWAS
A replication study of GWAS findings in migraine identifies association in a Swedish case–control sample
Gene, pathway and network frameworks to identify epistatic interactions of single nucleotide polymorphisms derived from GWAS data
Next-Gen GWAS: full 2D epistatic interaction maps retrieve part of missing heritability and improve phenotypic prediction
A random forest approach to the detection of epistatic interactions in case-control studies
Mapping epistatic quantitative trait loci
Systematic epistatic mapping of cellular processes
Missing value imputation for epistatic MAPs
OPE
Replication of LDL GWAs hits in PROSPER/PHASE as validation for future (pharmaco)genetic analyses
GWAS in IMIDs
Geographic differences in genetic susceptibility to IgA nephropathy: GWAS replication study and geospatial risk analysis
GWIS - model-free, fast and exhaustive search for epistatic interactions in case-control GWAS
A replication study of GWAS findings in migraine identifies association in a Swedish case–control sample
Gene, pathway and network frameworks to identify epistatic interactions of single nucleotide polymorphisms derived from GWAS data
Next-Gen GWAS: full 2D epistatic interaction maps retrieve part of missing heritability and improve phenotypic prediction
A random forest approach to the detection of epistatic interactions in case-control studies
Mapping epistatic quantitative trait loci
Systematic epistatic mapping of cellular processes
Missing value imputation for epistatic MAPs
OPE
Replication of LDL GWAs hits in PROSPER/PHASE as validation for future (pharmaco)genetic analyses
GWAS in IMIDs
Geographic differences in genetic susceptibility to IgA nephropathy: GWAS replication study and geospatial risk analysis
GWIS - model-free, fast and exhaustive search for epistatic interactions in case-control GWAS
A replication study of GWAS findings in migraine identifies association in a Swedish case–control sample
Gene, pathway and network frameworks to identify epistatic interactions of single nucleotide polymorphisms derived from GWAS data
Next-Gen GWAS: full 2D epistatic interaction maps retrieve part of missing heritability and improve phenotypic prediction
A random forest approach to the detection of epistatic interactions in case-control studies
Mapping epistatic quantitative trait loci
Systematic epistatic mapping of cellular processes
Missing value imputation for epistatic MAPs
OPE
Replication of LDL GWAs hits in PROSPER/PHASE as validation for future (pharmaco)genetic analyses
GWAS in IMIDs