Wnt3a disrupts GR-TEAD4-PPARγ2 positive circuits and cytoskeletal rearrangement in a β-catenin-dependent manner during early adipogenesis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-4889
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Wnt3a disrupts GR-TEAD4-PPARγ2 positive circuits and cytoskeletal rearrangement in a β-catenin-dependent manner during early adipogenesis ; volume:10 ; number:1 ; day:8 ; month:1 ; year:2019 ; pages:1-16 ; date:1.2019
Cell death & disease ; 10, Heft 1 (8.1.2019), 1-16, 1.2019
- Classification
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Biowissenschaften, Biologie
- Creator
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Park, Bongju
Chang, Soojeong
Lee, Gwan-Jun
Kang, Byeongsoo
Kim, Jong Kyoung
Park, Hyunsung
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1038/s41419-018-1249-7
- URN
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urn:nbn:de:101:1-2019021200335461905141
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:48 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Park, Bongju
- Chang, Soojeong
- Lee, Gwan-Jun
- Kang, Byeongsoo
- Kim, Jong Kyoung
- Park, Hyunsung
- SpringerLink (Online service)
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Knockdown of CDC20 promotes adipogenesis of bone marrow-derived stem cells by modulating β-catenin
Identification of mechanosensitive genes during skeletal development: alteration of genes associated with cytoskeletal rearrangement and cell signalling pathways
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Cytoskeletal rearrangement precedes nucleolar remodeling during adipogenesis
Cytoskeletal rearrangement during developmental motor axon remodeling
Glycolaldehyde disrupts insulin signaling and glucose uptake through adipogenesis
Toosendanin inhibits adipogenesis by activating Wnt/β-catenin signaling
A deep investigation into the adipogenesis mechanism: Profile of microRNAs regulating adipogenesis by modulating the canonical Wnt/β-catenin signaling pathway
IL-33 regulates adipogenesis via Wnt/β-catenin/PPAR-γ signaling pathway in preadipocytes
Knockdown of CDC20 promotes adipogenesis of bone marrow-derived stem cells by modulating β-catenin
Identification of mechanosensitive genes during skeletal development: alteration of genes associated with cytoskeletal rearrangement and cell signalling pathways
Azoramide, a novel regulator, favors adipogenesis against osteogenesis through inhibiting the GLP-1 receptor-PKA-β-catenin pathway
SLAMF8 regulates osteogenesis and adipogenesis of bone marrow mesenchymal stem cells via S100A6/Wnt/β-catenin signaling pathway
Hyperglycemia Altered the Fate of Cardiac Stem Cells to Adipogenesis through Inhibiting the β-Catenin/TCF-4 Pathway
Non-canonical WNT5A promotes cytoskeletal rearrangement and ITGAV clustering via JNK and ROCK to control the activation of latent TGFβ
Cytoskeletal rearrangement precedes nucleolar remodeling during adipogenesis
Cytoskeletal rearrangement during developmental motor axon remodeling
Glycolaldehyde disrupts insulin signaling and glucose uptake through adipogenesis
Toosendanin inhibits adipogenesis by activating Wnt/β-catenin signaling
A deep investigation into the adipogenesis mechanism: Profile of microRNAs regulating adipogenesis by modulating the canonical Wnt/β-catenin signaling pathway
IL-33 regulates adipogenesis via Wnt/β-catenin/PPAR-γ signaling pathway in preadipocytes
Knockdown of CDC20 promotes adipogenesis of bone marrow-derived stem cells by modulating β-catenin
Identification of mechanosensitive genes during skeletal development: alteration of genes associated with cytoskeletal rearrangement and cell signalling pathways
Azoramide, a novel regulator, favors adipogenesis against osteogenesis through inhibiting the GLP-1 receptor-PKA-β-catenin pathway
SLAMF8 regulates osteogenesis and adipogenesis of bone marrow mesenchymal stem cells via S100A6/Wnt/β-catenin signaling pathway
Hyperglycemia Altered the Fate of Cardiac Stem Cells to Adipogenesis through Inhibiting the β-Catenin/TCF-4 Pathway