Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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1 Online-Ressource.
- Sprache
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Englisch
- Erschienen in
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Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure ; volume:56 ; number:4 ; day:1 ; month:4 ; year:2024 ; pages:922-934 ; date:4.2024
Experimental & molecular medicine ; 56, Heft 4 (1.4.2024), 922-934, 4.2024
- Urheber
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Yoo, Youngbum
Yeon, MyeongHoon
Kim, Won-Kyung
Shin, Hyeon-Bin
Lee, Seung-Min
Yoon, Mee-Sup
Ro, Hyunju
Seo, Young-Kyo
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1038/s12276-024-01199-x
- URN
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urn:nbn:de:101:1-2407112112048.771667580537
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:52 MESZ
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Beteiligte
- Yoo, Youngbum
- Yeon, MyeongHoon
- Kim, Won-Kyung
- Shin, Hyeon-Bin
- Lee, Seung-Min
- Yoon, Mee-Sup
- Ro, Hyunju
- Seo, Young-Kyo
- SpringerLink (Online service)
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Cell stress molecules in the skeletal muscle of GNE myopathy
Stem cell activation in skeletal muscle regeneration
TEADs mediate pathway crosstalk during skeletal muscle regeneration
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Shp2 functions in postnatal skeletal muscle growth and regeneration
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PPARδ regulates satellite cell proliferation and skeletal muscle regeneration
Author Correction: Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
Tropomyosin 3 (TPM3) function in skeletal muscle and in myopathy
Cell stress molecules in the skeletal muscle of GNE myopathy
Stem cell activation in skeletal muscle regeneration
TEADs mediate pathway crosstalk during skeletal muscle regeneration
Bioengineered human skeletal muscle capable of functional regeneration
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Altered skeletal muscle (mitochondrial) properties in patients with mitochondrial DNA single deletion myopathy
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