Author Correction: Low oral dose of 4-methylumbelliferone reduces glial scar but is insufficient to induce functional recovery after spinal cord injury
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Author Correction: Low oral dose of 4-methylumbelliferone reduces glial scar but is insufficient to induce functional recovery after spinal cord injury ; volume:14 ; number:1 ; day:8 ; month:1 ; year:2024 ; pages:1-4 ; date:12.2024
Scientific reports ; 14, Heft 1 (8.1.2024), 1-4, 12.2024
- Creator
-
Štepánková, Kateřina
Chudíčková, Milada
Šimková, Zuzana
Martinez-Varea, Noelia
Kubinová, Šárka
Urdzíková, Lucia Machová
Jendelová, Pavla
Kwok, Jessica C. F.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41598-023-50043-1
- URN
-
urn:nbn:de:101:1-2024032209462904657869
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:49 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Štepánková, Kateřina
- Chudíčková, Milada
- Šimková, Zuzana
- Martinez-Varea, Noelia
- Kubinová, Šárka
- Urdzíková, Lucia Machová
- Jendelová, Pavla
- Kwok, Jessica C. F.
- SpringerLink (Online service)
Other Objects (12)
Low oral dose of 4-methylumbelliferone reduces glial scar but is insufficient to induce functional recovery after spinal cord injury
Propentofylline reduces glial scar development following gliotoxic damage in the rat brainstem
Anti-inflammatory polymer electrodes for glial scar treatment
New insights into glial scar formation after spinal cord injury
Engineering strategies towards overcoming bleeding and glial scar formation around neural probes
Dissecting glial scar formation by spatial point pattern and topological data analysis
The role of neural stem cells in regulating glial scar formation and repair
Glial senescence enhances α-synuclein pathology owing to its insufficient clearance caused by autophagy dysfunction
Insufficient difference
Insufficient difference
Autoantibodies against β1-adrenoceptor induce blood glucose enhancement and insulin insufficient via T lymphocytes
RGMa mediates reactive astrogliosis and glial scar formation through TGFβ1/Smad2/3 signaling after stroke
Low oral dose of 4-methylumbelliferone reduces glial scar but is insufficient to induce functional recovery after spinal cord injury
Propentofylline reduces glial scar development following gliotoxic damage in the rat brainstem
Anti-inflammatory polymer electrodes for glial scar treatment
New insights into glial scar formation after spinal cord injury
Engineering strategies towards overcoming bleeding and glial scar formation around neural probes
Dissecting glial scar formation by spatial point pattern and topological data analysis
The role of neural stem cells in regulating glial scar formation and repair
Glial senescence enhances α-synuclein pathology owing to its insufficient clearance caused by autophagy dysfunction
Insufficient difference
Insufficient difference
Autoantibodies against β1-adrenoceptor induce blood glucose enhancement and insulin insufficient via T lymphocytes
RGMa mediates reactive astrogliosis and glial scar formation through TGFβ1/Smad2/3 signaling after stroke
Low oral dose of 4-methylumbelliferone reduces glial scar but is insufficient to induce functional recovery after spinal cord injury
Propentofylline reduces glial scar development following gliotoxic damage in the rat brainstem
Anti-inflammatory polymer electrodes for glial scar treatment
New insights into glial scar formation after spinal cord injury
Engineering strategies towards overcoming bleeding and glial scar formation around neural probes
Dissecting glial scar formation by spatial point pattern and topological data analysis
The role of neural stem cells in regulating glial scar formation and repair
Glial senescence enhances α-synuclein pathology owing to its insufficient clearance caused by autophagy dysfunction
Insufficient difference
Insufficient difference
Autoantibodies against β1-adrenoceptor induce blood glucose enhancement and insulin insufficient via T lymphocytes