Toll-like receptors and NLRP3 inflammasome-dependent pathways in Parkinson’s disease: mechanisms and therapeutic implications
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1432-1459
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Toll-like receptors and NLRP3 inflammasome-dependent pathways in Parkinson’s disease: mechanisms and therapeutic implications ; day:3 ; month:12 ; year:2022 ; pages:1-15
Journal of neurology ; (3.12.2022), 1-15
- Creator
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Soraci, Luca
Gambuzza, Maria Elsa
Biscetti, Leonardo
Laganà, Pasqualina
Lo Russo, Carmela
Buda, Annamaria
Barresi, Giada
Corsonello, Andrea
Lattanzio, Fabrizia
Lorello, Giuseppe
Filippelli, Gianfranco
Marino, Silvia
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1007/s00415-022-11491-3
- URN
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urn:nbn:de:101:1-2023021421033963216209
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:56 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Soraci, Luca
- Gambuzza, Maria Elsa
- Biscetti, Leonardo
- Laganà, Pasqualina
- Lo Russo, Carmela
- Buda, Annamaria
- Barresi, Giada
- Corsonello, Andrea
- Lattanzio, Fabrizia
- Lorello, Giuseppe
- Filippelli, Gianfranco
- Marino, Silvia
- SpringerLink (Online service)
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Lack of genetic evidence for NLRP3 inflammasome involvement in Parkinson’s disease pathogenesis
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Pramipexole inhibits astrocytic NLRP3 inflammasome activation via Drd3-dependent autophagy in a mouse model of Parkinson’s disease
TREM2 Deficiency Aggravates NLRP3 Inflammasome Activation and Pyroptosis in MPTP-Induced Parkinson’s Disease Mice and LPS-Induced BV2 Cells
The circadian clock protein Rev-erbα provides neuroprotection and attenuates neuroinflammation against Parkinson’s disease via the microglial NLRP3 inflammasome
Systemic activation of NLRP3 inflammasome and plasma α-synuclein levels are correlated with motor severity and progression in Parkinson’s disease
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Plasma-borne indicators of inflammasome activity in Parkinson’s disease patients
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NLRP3 expression in mesencephalic neurons and characterization of a rare NLRP3 polymorphism associated with decreased risk of Parkinson’s disease
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Dopamine signaling modulates microglial NLRP3 inflammasome activation: implications for Parkinson’s disease
Lack of genetic evidence for NLRP3 inflammasome involvement in Parkinson’s disease pathogenesis
The role of Toll-like receptors and neuroinflammation in Parkinson’s disease
Pramipexole inhibits astrocytic NLRP3 inflammasome activation via Drd3-dependent autophagy in a mouse model of Parkinson’s disease
TREM2 Deficiency Aggravates NLRP3 Inflammasome Activation and Pyroptosis in MPTP-Induced Parkinson’s Disease Mice and LPS-Induced BV2 Cells
The circadian clock protein Rev-erbα provides neuroprotection and attenuates neuroinflammation against Parkinson’s disease via the microglial NLRP3 inflammasome
Systemic activation of NLRP3 inflammasome and plasma α-synuclein levels are correlated with motor severity and progression in Parkinson’s disease
Adenosine A2A receptors in Parkinson’s disease treatment
Plasma-borne indicators of inflammasome activity in Parkinson’s disease patients
Toll-like receptors contribute to non-canonical inflammasome-driven IL-1 release