Zinc finger nuclease-based double-strand breaks attenuate malaria parasites and reveal rare microhomology-mediated end joining
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
In: Genome Biology, 16 (2015), Nr. 249. pp. 1-18. ISSN 1474-760X
- Ereignis
-
Veröffentlichung
- (wo)
-
London
- (wer)
-
BioMed Central
- (wann)
-
2016
- Ereignis
-
Veröffentlichung
- (wo)
-
Heidelberg
- (wer)
-
Universitätsbibliothek Heidelberg
- (wann)
-
2016
- Urheber
-
Singer, Mirko
Marshall, Jennifer
Heiß, Kirsten
Mair, Gunnar R.
Grimm, Dirk
Müller, Ann-Kristin
Frischknecht, Friedrich
- DOI
-
10.1186/s13059-015-0811-1
- URN
-
urn:nbn:de:bsz:16-heidok-197927
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:58 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Singer, Mirko
- Marshall, Jennifer
- Heiß, Kirsten
- Mair, Gunnar R.
- Grimm, Dirk
- Müller, Ann-Kristin
- Frischknecht, Friedrich
- BioMed Central
- Universitätsbibliothek Heidelberg
Entstanden
- 2016
Ähnliche Objekte (12)
RNA-mediated double-strand break repair by end-joining mechanisms
5’-nuclease
High-complexity of DNA double-strand breaks is key for alternative end-joining choice
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1
Analysis of diverse double-strand break synapsis with Polλ reveals basis for unique substrate specificity in nonhomologous end-joining
The MRE11 nuclease promotes homologous recombination not only in DNA double-strand break resection but also in post-resection in human TK6 cells
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Parasites
Parasites
APE1 promotes non-homologous end joining by initiating DNA double-strand break formation and decreasing ubiquitination of artemis following oxidative genotoxic stress
A Click Chemistry‐Based Artificial Metallo‐Nuclease
RNA-mediated double-strand break repair by end-joining mechanisms
5’-nuclease
High-complexity of DNA double-strand breaks is key for alternative end-joining choice
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1
Analysis of diverse double-strand break synapsis with Polλ reveals basis for unique substrate specificity in nonhomologous end-joining
The MRE11 nuclease promotes homologous recombination not only in DNA double-strand break resection but also in post-resection in human TK6 cells
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Parasites
Parasites
APE1 promotes non-homologous end joining by initiating DNA double-strand break formation and decreasing ubiquitination of artemis following oxidative genotoxic stress
A Click Chemistry‐Based Artificial Metallo‐Nuclease
RNA-mediated double-strand break repair by end-joining mechanisms
5’-nuclease
High-complexity of DNA double-strand breaks is key for alternative end-joining choice
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1
Analysis of diverse double-strand break synapsis with Polλ reveals basis for unique substrate specificity in nonhomologous end-joining
The MRE11 nuclease promotes homologous recombination not only in DNA double-strand break resection but also in post-resection in human TK6 cells
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Parasites
Parasites
APE1 promotes non-homologous end joining by initiating DNA double-strand break formation and decreasing ubiquitination of artemis following oxidative genotoxic stress