- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Köln, Universität zu Köln, Diss., 2012
- Keyword
-
Hirnkrankheit
Neuropsychiatrie
Nervenkrankheit
- Event
-
Veröffentlichung
- (where)
-
Köln
- (who)
-
Universitäts- und Stadtbibliothek Köln
- (when)
-
2012
- Creator
- Contributor
-
Brüning, Jens C.
Krone, Wilhelm
- URN
-
urn:nbn:de:hbz:38-45720
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:50 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Moll, Lorna
- Brüning, Jens C.
- Krone, Wilhelm
- Universitäts- und Stadtbibliothek Köln
Time of origin
- 2012
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O-GlcNAcylation of FoxO1 mediates nucleoside diphosphate kinase B deficiency induced endothelial damage
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Mst1-mediated phosphorylation of FoxO1 and C/EBP-β stimulates cell-protective mechanisms in cardiomyocytes
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miR-92a-1-5p enriched prostate cancer extracellular vesicles regulate osteoclast function via MAPK1 and FoxO1
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Transcription factor FoxO1 regulates myoepithelial cell diversity and growth
HMGA1 is a novel transcriptional regulator of the FoxO1 gene
Cross-talk among HMGA1 and FoxO1 in control of nuclear insulin signaling
Mesenchymal stem cells promote pancreatic β-cell regeneration through downregulation of FoxO1 pathway
aPKC controls endothelial growth by modulating c-Myc via FoxO1 DNA-binding ability
O-GlcNAcylation of FoxO1 mediates nucleoside diphosphate kinase B deficiency induced endothelial damage
Liver FoxO1 overexpression is positively associated with the degree of liver injury in cirrhotic patients
Mst1-mediated phosphorylation of FoxO1 and C/EBP-β stimulates cell-protective mechanisms in cardiomyocytes
The Regulation of FoxO1 by Glucose-6-Phosphate Dehydrogenase and O-linked N-Acetyl-Glucosamine Modification
Human adipose and synovial-derived MSCs synergistically attenuate osteoarthritis by promoting chondrocyte autophagy through FoxO1 signaling
miR-92a-1-5p enriched prostate cancer extracellular vesicles regulate osteoclast function via MAPK1 and FoxO1
GlcNAcylation of FoxO1 is crucial for the upregulation of Angiopoietin 2 in NDPK B depleted endothelial cells
Transcription factor FoxO1 regulates myoepithelial cell diversity and growth
HMGA1 is a novel transcriptional regulator of the FoxO1 gene
Cross-talk among HMGA1 and FoxO1 in control of nuclear insulin signaling
Mesenchymal stem cells promote pancreatic β-cell regeneration through downregulation of FoxO1 pathway
aPKC controls endothelial growth by modulating c-Myc via FoxO1 DNA-binding ability
O-GlcNAcylation of FoxO1 mediates nucleoside diphosphate kinase B deficiency induced endothelial damage
Liver FoxO1 overexpression is positively associated with the degree of liver injury in cirrhotic patients
Mst1-mediated phosphorylation of FoxO1 and C/EBP-β stimulates cell-protective mechanisms in cardiomyocytes
The Regulation of FoxO1 by Glucose-6-Phosphate Dehydrogenase and O-linked N-Acetyl-Glucosamine Modification
Human adipose and synovial-derived MSCs synergistically attenuate osteoarthritis by promoting chondrocyte autophagy through FoxO1 signaling
miR-92a-1-5p enriched prostate cancer extracellular vesicles regulate osteoclast function via MAPK1 and FoxO1