Predictive value of the SLC22A18 protein expression in glioblastoma patients receiving temozolomide therapy
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1479-5876
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Predictive value of the SLC22A18 protein expression in glioblastoma patients receiving temozolomide therapy ; volume:11 ; number:1 ; day:20 ; month:3 ; year:2013 ; pages:1-8 ; date:12.2013
Journal of translational medicine ; 11, Heft 1 (20.3.2013), 1-8, 12.2013
- Creator
-
Chu, Sheng-Hua
- Contributor
-
Ma, Yan-Bin
Feng, Dong-Fu
Li, Zhi-Qiang
Jiang, Pu-Cha
SpringerLink (Online service)
- DOI
-
10.1186/1479-5876-11-69
- URN
-
urn:nbn:de:1111-2016041444975
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:56 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Chu, Sheng-Hua
- Ma, Yan-Bin
- Feng, Dong-Fu
- Li, Zhi-Qiang
- Jiang, Pu-Cha
- SpringerLink (Online service)
Other Objects (12)
Accelerated hyperfractionation plus temozolomide in glioblastoma
Predictive value of MRI features on glioblastoma
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Predictive value of tumor recurrence using urinary vascular endothelial factor levels in patients receiving radiation therapy for Glioblastoma Multiforme (GBM)
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Combined effects of niclosamide and temozolomide against human glioblastoma tumorspheres
One-carbon-mediated purine synthesis underlies temozolomide resistance in glioblastoma
Inhibition of gap junctions sensitizes primary glioblastoma cells for temozolomide
Glioblastoma stem cells deliver ABCB4 transcribed by ATF3 via exosomes conferring glioblastoma resistance to temozolomide
Markedly divergent effects of Ouabain on a Temozolomide-resistant (T98G) vs. a Temozolomide-sensitive (LN229) Glioblastoma cell line
Accelerated hyperfractionation plus temozolomide in glioblastoma
Predictive value of MRI features on glioblastoma
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Predictive value of tumor recurrence using urinary vascular endothelial factor levels in patients receiving radiation therapy for Glioblastoma Multiforme (GBM)
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Combined effects of niclosamide and temozolomide against human glioblastoma tumorspheres
One-carbon-mediated purine synthesis underlies temozolomide resistance in glioblastoma
Inhibition of gap junctions sensitizes primary glioblastoma cells for temozolomide
Glioblastoma stem cells deliver ABCB4 transcribed by ATF3 via exosomes conferring glioblastoma resistance to temozolomide
Markedly divergent effects of Ouabain on a Temozolomide-resistant (T98G) vs. a Temozolomide-sensitive (LN229) Glioblastoma cell line
Accelerated hyperfractionation plus temozolomide in glioblastoma
Predictive value of MRI features on glioblastoma
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Predictive value of tumor recurrence using urinary vascular endothelial factor levels in patients receiving radiation therapy for Glioblastoma Multiforme (GBM)
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Combined effects of niclosamide and temozolomide against human glioblastoma tumorspheres
One-carbon-mediated purine synthesis underlies temozolomide resistance in glioblastoma
Inhibition of gap junctions sensitizes primary glioblastoma cells for temozolomide
Glioblastoma stem cells deliver ABCB4 transcribed by ATF3 via exosomes conferring glioblastoma resistance to temozolomide