Loss of SIM2s inhibits RAD51 binding and leads to unresolved replication stress
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1465-542X
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Loss of SIM2s inhibits RAD51 binding and leads to unresolved replication stress ; volume:21 ; number:1 ; day:27 ; month:11 ; year:2019 ; pages:1-14 ; date:12.2019
Breast cancer research ; 21, Heft 1 (27.11.2019), 1-14, 12.2019
- Urheber
-
Pearson, Scott J.
Elswood, Jessica
Barhoumi, Rola
Ming-Whitfield, Brittini
Rijnkels, Monique
Porter, Weston W.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s13058-019-1207-z
- URN
-
urn:nbn:de:101:1-2020032221215343925208
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:58 MESZ
Datenpartner
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Beteiligte
- Pearson, Scott J.
- Elswood, Jessica
- Barhoumi, Rola
- Ming-Whitfield, Brittini
- Rijnkels, Monique
- Porter, Weston W.
- SpringerLink (Online service)
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Synthetic lethality by PARP inhibitors: new mechanism uncovered based on unresolved transcription-replication conflicts
GRB2 stabilizes RAD51 at reversed replication forks suppressing genomic instability and innate immunity against cancer
Sister chromatid exchanges induced by perturbed replication can form independently of BRCA1, BRCA2 and RAD51
Aprotinin inhibits SARS-CoV-2 replication
PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation
FIGNL1 AAA+ ATPase remodels RAD51 and DMC1 filaments in pre-meiotic DNA replication and meiotic recombination
2-Hydroxy-3-methylanthraquinone inhibits homologous recombination repair in osteosarcoma through the MYC-CHK1-RAD51 axis
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Heterologous SH3-p85β inhibits influenza A virus replication
ZNF148 inhibits HBV replication by downregulating RXRα transcription
Glycyrrhiza polysaccharides inhibits PRRSV replication
FLIP(C1orf112)-FIGNL1 complex regulates RAD51 chromatin association to promote viability after replication stress
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GRB2 stabilizes RAD51 at reversed replication forks suppressing genomic instability and innate immunity against cancer
Sister chromatid exchanges induced by perturbed replication can form independently of BRCA1, BRCA2 and RAD51
Aprotinin inhibits SARS-CoV-2 replication
PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation
FIGNL1 AAA+ ATPase remodels RAD51 and DMC1 filaments in pre-meiotic DNA replication and meiotic recombination
2-Hydroxy-3-methylanthraquinone inhibits homologous recombination repair in osteosarcoma through the MYC-CHK1-RAD51 axis
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Heterologous SH3-p85β inhibits influenza A virus replication