Hochschulschrift
Role of BRD4 and histone acetylation in estrogen receptor-positive breast cancers
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Göttingen, Georg-August Universität, Diss., 2015
- Keyword
-
Krebs
Onkologie
- Event
-
Veröffentlichung
- (where)
-
Göttingen
- (who)
-
Niedersächsische Staats- und Universitätsbibliothek Göttingen
- (when)
-
2015
- Creator
-
Nagarajan, Sankari
- Contributor
-
Johnsen, Steven
Kube, Dieter
Shcherbata, Halyna
- Handle
-
11858/00-1735-0000-0023-9615-4
- URN
-
urn:nbn:de:gbv:7-11858/00-1735-0000-0023-9615-4-4
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:44 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Nagarajan, Sankari
- Johnsen, Steven
- Kube, Dieter
- Shcherbata, Halyna
- Niedersächsische Staats- und Universitätsbibliothek Göttingen
Time of origin
- 2015
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Estrogen and breast cancer
Proliferation and estrogen signaling can distinguish patients at risk for early versus late relapse among estrogen receptor positive breast cancers
Nitric oxide affects histone acetylation by inhibiting histone deacetylase activity in Arabidopsis
T-cell metagene predicts a favorable prognosis in estrogen receptor-negative and HER2-positive breast cancers
Histone acetylation and histone acetyltransferases show significant alterations in human abdominal aortic aneurysm
Histone acetylation and expression of corresponding histone actyltransferases in human abdominal aortic aneurysm
Histone chaperone exploits intrinsic disorder to switch acetylation specificity
A new approach to the function of histone acetylation
Involvement of histone acetylation in neuroprotection against brain ischemic injury
Estrogen receptor gene (ESR1) amplification occurs rarely in ovarian cancers