Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer’s Pathogenesis

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer’s Pathogenesis ; day:24 ; month:10 ; year:2024 ; pages:1-29
Molecular neurobiology ; (24.10.2024), 1-29

Creator: Almeida, Zaida L.
Vaz, Daniela C.
Brito, Rui M. M.

Contributor: SpringerLink (Online service)

DOI: 10.1007/s12035-024-04543-4

URN: urn:nbn:de:101:1-2501132110416.876886458182

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:23 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Almeida, Zaida L.
Vaz, Daniela C.
Brito, Rui M. M.
SpringerLink (Online service)

Other Objects (12)

Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease

Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease

Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit

Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission

Peripheral blood amyloid-β involved in the pathogenesis of Alzheimer’s disease via impacting on peripheral innate immune cells

Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease

A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease

Astrocyte biomarker signatures of amyloid-β and tau pathologies in Alzheimer’s disease

Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease

Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease

Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit

Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission

Peripheral blood amyloid-β involved in the pathogenesis of Alzheimer’s disease via impacting on peripheral innate immune cells

Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease

A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease

Astrocyte biomarker signatures of amyloid-β and tau pathologies in Alzheimer’s disease

Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease

Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease

Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit

Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission

Peripheral blood amyloid-β involved in the pathogenesis of Alzheimer’s disease via impacting on peripheral innate immune cells

Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease

A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease

Astrocyte biomarker signatures of amyloid-β and tau pathologies in Alzheimer’s disease

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer’s Pathogenesis

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease

Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease

Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit

Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission

Peripheral blood amyloid-β involved in the pathogenesis of Alzheimer’s disease via impacting on peripheral innate immune cells

Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease

A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease

Astrocyte biomarker signatures of amyloid-β and tau pathologies in Alzheimer’s disease

Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease

Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease

Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit

Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission

Peripheral blood amyloid-β involved in the pathogenesis of Alzheimer’s disease via impacting on peripheral innate immune cells

Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease

A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease

Astrocyte biomarker signatures of amyloid-β and tau pathologies in Alzheimer’s disease

Nanoscale structure of amyloid-β plaques in Alzheimer’s disease

Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease

Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease

Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease

Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit

Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease

Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission

Peripheral blood amyloid-β involved in the pathogenesis of Alzheimer’s disease via impacting on peripheral innate immune cells

Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease

A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease

Astrocyte biomarker signatures of amyloid-β and tau pathologies in Alzheimer’s disease

Related objects

Reset password