Gasdermin D mediates endoplasmic reticulum stress via FAM134B to regulate cardiomyocyte autophagy and apoptosis in doxorubicin-induced cardiotoxicity

to related objects

Location
Deutsche Nationalbibliothek Frankfurt am Main
ISSN
2041-4889
Extent
Online-Ressource
Language
Englisch
Notes
online resource.

Bibliographic citation
Gasdermin D mediates endoplasmic reticulum stress via FAM134B to regulate cardiomyocyte autophagy and apoptosis in doxorubicin-induced cardiotoxicity ; volume:13 ; number:10 ; day:26 ; month:10 ; year:2022 ; pages:1-14 ; date:10.2022
Cell death & disease ; 13, Heft 10 (26.10.2022), 1-14, 10.2022

Creator
Qu, Ya’nan
Gao, Rifeng
Wei, Xiang
Sun, Xiaolei
Yang, Kun
Shi, Huairui
Gao, Yang
Hu, Shiyu
Wang, Yiwen
Yang, Ji’e
Sun, Aijun
Zhang, Feng
Ge, Junbo
Contributor
SpringerLink (Online service)

DOI
10.1038/s41419-022-05333-3
URN
urn:nbn:de:101:1-2023020409161943266598
Rights
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
Last update
14.08.2025, 10:54 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Show original at data provider

Associated

  • Qu, Ya’nan
  • Gao, Rifeng
  • Wei, Xiang
  • Sun, Xiaolei
  • Yang, Kun
  • Shi, Huairui
  • Gao, Yang
  • Hu, Shiyu
  • Wang, Yiwen
  • Yang, Ji’e
  • Sun, Aijun
  • Zhang, Feng
  • Ge, Junbo
  • SpringerLink (Online service)

Other Objects (12)

Correction to: Gasdermin D mediates endoplasmic reticulum stress via FAM134B to regulate cardiomyocyte autophagy and apoptosis in doxorubicin-induced cardiotoxicity

Correction to: Gasdermin D mediates endoplasmic reticulum stress via FAM134B to regulate cardiomyocyte autophagy and apoptosis in doxorubicin-induced cardiotoxicity

Cardiomyocyte-specific disruption of Cathepsin K protects against doxorubicin-induced cardiotoxicity

Cardiomyocyte-specific disruption of Cathepsin K protects against doxorubicin-induced cardiotoxicity

Rifampicin efficacy against doxorubicin-induced cardiotoxicity in mice

Rifampicin efficacy against doxorubicin-induced cardiotoxicity in mice

Neutrophil extracellular traps mediate cardiomyocyte ferroptosis via the Hippo–Yap pathway to exacerbate doxorubicin-induced cardiotoxicity

Neutrophil extracellular traps mediate cardiomyocyte ferroptosis via the Hippo–Yap pathway to exacerbate doxorubicin-induced cardiotoxicity

Diazoxide protects against doxorubicin-induced cardiotoxicity in the rat

Diazoxide protects against doxorubicin-induced cardiotoxicity in the rat

Doxorubicin-induced cardiotoxicity in adult Indian patients on chemotherapy

Doxorubicin-induced cardiotoxicity in adult Indian patients on chemotherapy

Protective effect of silymarin on doxorubicin-induced cardiotoxicity in rats

Protective effect of silymarin on doxorubicin-induced cardiotoxicity in rats

Sex Differences in Doxorubicin‐Induced Cardiotoxicity: Insights from Transcriptome Analysis

Sex Differences in Doxorubicin‐Induced Cardiotoxicity: Insights from Transcriptome Analysis

Tripartite motif 25 ameliorates doxorubicin-induced cardiotoxicity by degrading p85α

Tripartite motif 25 ameliorates doxorubicin-induced cardiotoxicity by degrading p85α

The significance of the apelinergic system in doxorubicin-induced cardiotoxicity

The significance of the apelinergic system in doxorubicin-induced cardiotoxicity

The Role of AMPK Activation for Cardioprotection in Doxorubicin-Induced Cardiotoxicity

The Role of AMPK Activation for Cardioprotection in Doxorubicin-Induced Cardiotoxicity

Shikonin alleviates doxorubicin-induced cardiotoxicity via Mst1/Nrf2 pathway in mice

Shikonin alleviates doxorubicin-induced cardiotoxicity via Mst1/Nrf2 pathway in mice

Related objects