C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial–mesenchymal transition in COPD mice

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2466

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial–mesenchymal transition in COPD mice ; volume:21 ; number:1 ; day:18 ; month:11 ; year:2021 ; pages:1-11 ; date:12.2021
BMC pulmonary medicine ; 21, Heft 1 (18.11.2021), 1-11, 12.2021

Creator: Chu, Shuyuan
Ma, Libing
Wu, Yashan
Zhao, Xiaoli
Xiao, Bo
Pan, Qilu

Contributor: SpringerLink (Online service)

DOI: 10.1186/s12890-021-01738-6

URN: urn:nbn:de:101:1-2022012821211084604217

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:31 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Chu, Shuyuan
Ma, Libing
Wu, Yashan
Zhao, Xiaoli
Xiao, Bo
Pan, Qilu
SpringerLink (Online service)

Other Objects (12)

NF-kappaB mediates the survival of human bronchial epithelial cells exposed to cigarette smoke extract

Cigarette and IL-17A synergistically induce bronchial epithelial-mesenchymal transition via activating IL-17R/NF-κB signaling

Cigarette smoke and electronic cigarettes differentially activate bronchial epithelial cells

Bronchial microbiome of severe COPD patients colonised by Pseudomonas aeruginosa

Bronchial extracellular matrix from COPD patients induces altered gene expression in repopulated primary human bronchial epithelial cells

Cigarette smoke extract stimulates bronchial epithelial cells to undergo a SUMOylation turnover

CD147 increases mucus secretion induced by cigarette smoke in COPD

Early transcriptional responses of bronchial epithelial cells to whole cigarette smoke mirror those of in-vivo exposed human bronchial mucosa

Smoking cessation and bronchial epithelial remodelling in COPD: a cross-sectional study

Nasal gene expression differentiates COPD from controls and overlaps bronchial gene expression

Progesterone (P4) ameliorates cigarette smoke-induced chronic obstructive pulmonary disease (COPD)

Mechanism of cigarette smoke condensate-induced acute inflammatory response in human bronchial epithelial cells

NF-kappaB mediates the survival of human bronchial epithelial cells exposed to cigarette smoke extract

Cigarette and IL-17A synergistically induce bronchial epithelial-mesenchymal transition via activating IL-17R/NF-κB signaling

Cigarette smoke and electronic cigarettes differentially activate bronchial epithelial cells

Bronchial microbiome of severe COPD patients colonised by Pseudomonas aeruginosa

Bronchial extracellular matrix from COPD patients induces altered gene expression in repopulated primary human bronchial epithelial cells

Cigarette smoke extract stimulates bronchial epithelial cells to undergo a SUMOylation turnover

CD147 increases mucus secretion induced by cigarette smoke in COPD

Early transcriptional responses of bronchial epithelial cells to whole cigarette smoke mirror those of in-vivo exposed human bronchial mucosa

Smoking cessation and bronchial epithelial remodelling in COPD: a cross-sectional study

Nasal gene expression differentiates COPD from controls and overlaps bronchial gene expression

Progesterone (P4) ameliorates cigarette smoke-induced chronic obstructive pulmonary disease (COPD)

Mechanism of cigarette smoke condensate-induced acute inflammatory response in human bronchial epithelial cells

NF-kappaB mediates the survival of human bronchial epithelial cells exposed to cigarette smoke extract

Cigarette and IL-17A synergistically induce bronchial epithelial-mesenchymal transition via activating IL-17R/NF-κB signaling

Cigarette smoke and electronic cigarettes differentially activate bronchial epithelial cells

Bronchial microbiome of severe COPD patients colonised by Pseudomonas aeruginosa

Bronchial extracellular matrix from COPD patients induces altered gene expression in repopulated primary human bronchial epithelial cells

Cigarette smoke extract stimulates bronchial epithelial cells to undergo a SUMOylation turnover

CD147 increases mucus secretion induced by cigarette smoke in COPD

Early transcriptional responses of bronchial epithelial cells to whole cigarette smoke mirror those of in-vivo exposed human bronchial mucosa

Smoking cessation and bronchial epithelial remodelling in COPD: a cross-sectional study

Nasal gene expression differentiates COPD from controls and overlaps bronchial gene expression

Progesterone (P4) ameliorates cigarette smoke-induced chronic obstructive pulmonary disease (COPD)

Mechanism of cigarette smoke condensate-induced acute inflammatory response in human bronchial epithelial cells

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial–mesenchymal transition in COPD mice

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

NF-kappaB mediates the survival of human bronchial epithelial cells exposed to cigarette smoke extract

Cigarette and IL-17A synergistically induce bronchial epithelial-mesenchymal transition via activating IL-17R/NF-κB signaling

Cigarette smoke and electronic cigarettes differentially activate bronchial epithelial cells

Bronchial microbiome of severe COPD patients colonised by Pseudomonas aeruginosa

Bronchial extracellular matrix from COPD patients induces altered gene expression in repopulated primary human bronchial epithelial cells

Cigarette smoke extract stimulates bronchial epithelial cells to undergo a SUMOylation turnover

CD147 increases mucus secretion induced by cigarette smoke in COPD

Early transcriptional responses of bronchial epithelial cells to whole cigarette smoke mirror those of in-vivo exposed human bronchial mucosa

Smoking cessation and bronchial epithelial remodelling in COPD: a cross-sectional study

Nasal gene expression differentiates COPD from controls and overlaps bronchial gene expression

Progesterone (P4) ameliorates cigarette smoke-induced chronic obstructive pulmonary disease (COPD)

Mechanism of cigarette smoke condensate-induced acute inflammatory response in human bronchial epithelial cells

NF-kappaB mediates the survival of human bronchial epithelial cells exposed to cigarette smoke extract

Cigarette and IL-17A synergistically induce bronchial epithelial-mesenchymal transition via activating IL-17R/NF-κB signaling

Cigarette smoke and electronic cigarettes differentially activate bronchial epithelial cells

Bronchial microbiome of severe COPD patients colonised by Pseudomonas aeruginosa

Bronchial extracellular matrix from COPD patients induces altered gene expression in repopulated primary human bronchial epithelial cells

Cigarette smoke extract stimulates bronchial epithelial cells to undergo a SUMOylation turnover

CD147 increases mucus secretion induced by cigarette smoke in COPD

Early transcriptional responses of bronchial epithelial cells to whole cigarette smoke mirror those of in-vivo exposed human bronchial mucosa

Smoking cessation and bronchial epithelial remodelling in COPD: a cross-sectional study

Nasal gene expression differentiates COPD from controls and overlaps bronchial gene expression

Progesterone (P4) ameliorates cigarette smoke-induced chronic obstructive pulmonary disease (COPD)

Mechanism of cigarette smoke condensate-induced acute inflammatory response in human bronchial epithelial cells

NF-kappaB mediates the survival of human bronchial epithelial cells exposed to cigarette smoke extract

Cigarette and IL-17A synergistically induce bronchial epithelial-mesenchymal transition via activating IL-17R/NF-κB signaling

Cigarette smoke and electronic cigarettes differentially activate bronchial epithelial cells

Bronchial microbiome of severe COPD patients colonised by Pseudomonas aeruginosa

Bronchial extracellular matrix from COPD patients induces altered gene expression in repopulated primary human bronchial epithelial cells

Cigarette smoke extract stimulates bronchial epithelial cells to undergo a SUMOylation turnover

CD147 increases mucus secretion induced by cigarette smoke in COPD

Early transcriptional responses of bronchial epithelial cells to whole cigarette smoke mirror those of in-vivo exposed human bronchial mucosa

Smoking cessation and bronchial epithelial remodelling in COPD: a cross-sectional study

Nasal gene expression differentiates COPD from controls and overlaps bronchial gene expression

Progesterone (P4) ameliorates cigarette smoke-induced chronic obstructive pulmonary disease (COPD)

Mechanism of cigarette smoke condensate-induced acute inflammatory response in human bronchial epithelial cells

Related objects

Reset password