CCDC88A, a prognostic factor for human pancreatic cancers, promotes the motility and invasiveness of pancreatic cancer cells
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-9966
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
CCDC88A, a prognostic factor for human pancreatic cancers, promotes the motility and invasiveness of pancreatic cancer cells ; volume:35 ; number:1 ; day:5 ; month:12 ; year:2016 ; pages:1-17 ; date:12.2016
Journal of experimental & clinical cancer research ; 35, Heft 1 (5.12.2016), 1-17, 12.2016
- Urheber
-
Tanouchi, Aki
- Beteiligte Personen und Organisationen
-
Taniuchi, Keisuke
Furihata, Mutsuo
Naganuma, Seiji
Dabanaka, Ken
Kimura, Masashi
Watanabe, Ryohei
Kohsaki, Takuhiro
Shimizu, Takahiro
Saito, Motoaki
Hanazaki, Kazuhiro
Saibara, Toshiji
SpringerLink (Online service)
- DOI
-
10.1186/s13046-016-0466-0
- URN
-
urn:nbn:de:1111-201701095892
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:49 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Tanouchi, Aki
- Taniuchi, Keisuke
- Furihata, Mutsuo
- Naganuma, Seiji
- Dabanaka, Ken
- Kimura, Masashi
- Watanabe, Ryohei
- Kohsaki, Takuhiro
- Shimizu, Takahiro
- Saito, Motoaki
- Hanazaki, Kazuhiro
- Saibara, Toshiji
- SpringerLink (Online service)
Ähnliche Objekte (12)
CCR5/CCL5 axis interaction promotes migratory and invasiveness of pancreatic cancer cells
Corticotropin Releasing Factor promotes breast cancer cell motility and invasiveness
Linc00462 promotes pancreatic cancer invasiveness through the miR-665/TGFBR1-TGFBR2/SMAD2/3 pathway
Original Scientific Reports: Clinicopathological Findings of Remnant Pancreatic Cancers in Survivors Following Curative Resections of Pancreatic Cancers
Host Matrix Modulation by Tumor Exosomal CD44v6 and Proteases Promotes Motility and Invasiveness
Melanoma Inhibitory Activity (MIA) increases the invasiveness of pancreatic cancer cells
Engrailed‐1 Promotes Pancreatic Cancer Metastasis
CHD7 promotes glioblastoma cell motility and invasiveness through transcriptional modulation of an invasion signature
PTEN regulates invasiveness in pancreatic neuroendocrine tumors through DUSP19-mediated VEGFR3 dephosphorylation
Pancreatic cancers depend on a non-canonical glutamine metabolism pathway
Infected Pancreatic Necrosis Mimicking Pancreatic Cancer
Secretion of fibronectin by human pancreatic stellate cells promotes chemoresistance to gemcitabine in pancreatic cancer cells
CCR5/CCL5 axis interaction promotes migratory and invasiveness of pancreatic cancer cells
Corticotropin Releasing Factor promotes breast cancer cell motility and invasiveness
Linc00462 promotes pancreatic cancer invasiveness through the miR-665/TGFBR1-TGFBR2/SMAD2/3 pathway
Original Scientific Reports: Clinicopathological Findings of Remnant Pancreatic Cancers in Survivors Following Curative Resections of Pancreatic Cancers
Host Matrix Modulation by Tumor Exosomal CD44v6 and Proteases Promotes Motility and Invasiveness
Melanoma Inhibitory Activity (MIA) increases the invasiveness of pancreatic cancer cells
Engrailed‐1 Promotes Pancreatic Cancer Metastasis
CHD7 promotes glioblastoma cell motility and invasiveness through transcriptional modulation of an invasion signature
PTEN regulates invasiveness in pancreatic neuroendocrine tumors through DUSP19-mediated VEGFR3 dephosphorylation
Pancreatic cancers depend on a non-canonical glutamine metabolism pathway
Infected Pancreatic Necrosis Mimicking Pancreatic Cancer
Secretion of fibronectin by human pancreatic stellate cells promotes chemoresistance to gemcitabine in pancreatic cancer cells
CCR5/CCL5 axis interaction promotes migratory and invasiveness of pancreatic cancer cells
Corticotropin Releasing Factor promotes breast cancer cell motility and invasiveness
Linc00462 promotes pancreatic cancer invasiveness through the miR-665/TGFBR1-TGFBR2/SMAD2/3 pathway
Original Scientific Reports: Clinicopathological Findings of Remnant Pancreatic Cancers in Survivors Following Curative Resections of Pancreatic Cancers
Host Matrix Modulation by Tumor Exosomal CD44v6 and Proteases Promotes Motility and Invasiveness
Melanoma Inhibitory Activity (MIA) increases the invasiveness of pancreatic cancer cells
Engrailed‐1 Promotes Pancreatic Cancer Metastasis
CHD7 promotes glioblastoma cell motility and invasiveness through transcriptional modulation of an invasion signature
PTEN regulates invasiveness in pancreatic neuroendocrine tumors through DUSP19-mediated VEGFR3 dephosphorylation
Pancreatic cancers depend on a non-canonical glutamine metabolism pathway
Infected Pancreatic Necrosis Mimicking Pancreatic Cancer