Stemness of Cancer: A Study of Triple-negative Breast Cancer From a Neuroscience Perspective
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Stemness of Cancer: A Study of Triple-negative Breast Cancer From a Neuroscience Perspective ; day:12 ; month:11 ; year:2024 ; pages:1-14
Stem cell reviews and reports ; (12.11.2024), 1-14
- Creator
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Djamgoz, Mustafa B. A.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s12015-024-10809-0
- URN
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urn:nbn:de:101:1-2501292108249.252033397407
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:36 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Djamgoz, Mustafa B. A.
- SpringerLink (Online service)
Other Objects (12)
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METTL14 suppresses the expression of YAP1 and the stemness of triple-negative breast cancer
NAC1 promotes stemness and regulates myeloid-derived cell status in triple-negative breast cancer
Chemotherapeutic Stress Influences Epithelial-Mesenchymal Transition and Stemness in Cancer Stem Cells of Triple-Negative Breast Cancer
Tyrosine kinase SRC-induced YAP1-KLF5 module regulates cancer stemness and metastasis in triple-negative breast cancer
Characterizing triple-negative breast cancer tumors
Triple-Negative Breast Cancer: Multimodality Appearance
Loss of OVOL2 in Triple‐Negative Breast Cancer Promotes Fatty Acid Oxidation Fueling Stemness Characteristics
Correction: Nac1 promotes stemness and regulates myeloid‑derived cell status in triple‑negative breast cancer
Long non-coding RNA NEAT1 confers oncogenic role in triple-negative breast cancer through modulating chemoresistance and cancer stemness
Prognostic model based on tumor stemness genes for triple-negative breast cancer
THOC2 and THOC5 Regulate Stemness and Radioresistance in Triple‐Negative Breast Cancer
Metastatic Triple-Negative Breast Cancer
METTL14 suppresses the expression of YAP1 and the stemness of triple-negative breast cancer
NAC1 promotes stemness and regulates myeloid-derived cell status in triple-negative breast cancer
Chemotherapeutic Stress Influences Epithelial-Mesenchymal Transition and Stemness in Cancer Stem Cells of Triple-Negative Breast Cancer
Tyrosine kinase SRC-induced YAP1-KLF5 module regulates cancer stemness and metastasis in triple-negative breast cancer
Characterizing triple-negative breast cancer tumors
Triple-Negative Breast Cancer: Multimodality Appearance
Loss of OVOL2 in Triple‐Negative Breast Cancer Promotes Fatty Acid Oxidation Fueling Stemness Characteristics
Correction: Nac1 promotes stemness and regulates myeloid‑derived cell status in triple‑negative breast cancer
Long non-coding RNA NEAT1 confers oncogenic role in triple-negative breast cancer through modulating chemoresistance and cancer stemness
Prognostic model based on tumor stemness genes for triple-negative breast cancer
THOC2 and THOC5 Regulate Stemness and Radioresistance in Triple‐Negative Breast Cancer
Metastatic Triple-Negative Breast Cancer
METTL14 suppresses the expression of YAP1 and the stemness of triple-negative breast cancer
NAC1 promotes stemness and regulates myeloid-derived cell status in triple-negative breast cancer
Chemotherapeutic Stress Influences Epithelial-Mesenchymal Transition and Stemness in Cancer Stem Cells of Triple-Negative Breast Cancer
Tyrosine kinase SRC-induced YAP1-KLF5 module regulates cancer stemness and metastasis in triple-negative breast cancer
Characterizing triple-negative breast cancer tumors
Triple-Negative Breast Cancer: Multimodality Appearance
Loss of OVOL2 in Triple‐Negative Breast Cancer Promotes Fatty Acid Oxidation Fueling Stemness Characteristics
Correction: Nac1 promotes stemness and regulates myeloid‑derived cell status in triple‑negative breast cancer