- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-3701
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
The first structure of HIV-1 gp120 with CD4 and CCR5 receptors ; volume:9 ; number:1 ; day:3 ; month:1 ; year:2019 ; pages:1-3 ; date:12.2019
Cell & bioscience ; 9, Heft 1 (3.1.2019), 1-3, 12.2019
- Klassifikation
-
Biowissenschaften, Biologie
- Urheber
-
Guan, Yongjun
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s13578-018-0267-6
- URN
-
urn:nbn:de:101:1-2019020803101117561507
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 11:01 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Guan, Yongjun
- SpringerLink (Online service)
Ähnliche Objekte (12)
Blockade of chemokine-induced signalling inhibits CCR5-dependent HIV infection in vitro without blocking gp120/CCR5 interaction
Antibodies to the CD4-binding site of HIV-1 gp120 suppress gp120-specific CD4 T cell response while enhancing antibody response
Allosteric induction of the CD4-bound conformation of HIV-1 Gp120
Genome editing of the HIV co-receptors CCR5 and CXCR4 by CRISPR-Cas9 protects CD4+ T cells from HIV-1 infection
Conformational alterations in the CD4 binding cavity of HIV-1 gp120 influencing gp120-CD4 interactions and fusogenicity of HIV-1 envelopes derived from brain and other tissues
Characterization of gp120 and Its Single-chain Derivatives, gp120-CD4 and gp120-M9: Implications for Targeting the CD4i Epitope in HIV-1 Vaccine Design
HIV-1 gp120 receptor on CD4-negative brain cells activates a tyrosine kinase
Recombinant soluble CCR5 AND CXCR4 chemokine receptors as anti-HIV drug targets
IFNα induces CCR5 in CD4+ T cells of HIV patients causing pathogenic elevation
Modellierung der Interaktion des humanen Korezeptor CCR5 mit der V3-Loop des Glycoproteins gp120 aus dem HIV-1
Das "Strukturschalterphänomen" der CD4-Bindungsdomäne des HIV-1-Oberflächenproteins gp120 und dessen exogene Inhibition
Effect of site-specific de-glycosylation on HIV gp120-specific CD4 T cell responses
Blockade of chemokine-induced signalling inhibits CCR5-dependent HIV infection in vitro without blocking gp120/CCR5 interaction
Antibodies to the CD4-binding site of HIV-1 gp120 suppress gp120-specific CD4 T cell response while enhancing antibody response
Allosteric induction of the CD4-bound conformation of HIV-1 Gp120
Genome editing of the HIV co-receptors CCR5 and CXCR4 by CRISPR-Cas9 protects CD4+ T cells from HIV-1 infection
Conformational alterations in the CD4 binding cavity of HIV-1 gp120 influencing gp120-CD4 interactions and fusogenicity of HIV-1 envelopes derived from brain and other tissues
Characterization of gp120 and Its Single-chain Derivatives, gp120-CD4 and gp120-M9: Implications for Targeting the CD4i Epitope in HIV-1 Vaccine Design
HIV-1 gp120 receptor on CD4-negative brain cells activates a tyrosine kinase
Recombinant soluble CCR5 AND CXCR4 chemokine receptors as anti-HIV drug targets
IFNα induces CCR5 in CD4+ T cells of HIV patients causing pathogenic elevation
Modellierung der Interaktion des humanen Korezeptor CCR5 mit der V3-Loop des Glycoproteins gp120 aus dem HIV-1
Das "Strukturschalterphänomen" der CD4-Bindungsdomäne des HIV-1-Oberflächenproteins gp120 und dessen exogene Inhibition
Effect of site-specific de-glycosylation on HIV gp120-specific CD4 T cell responses
Blockade of chemokine-induced signalling inhibits CCR5-dependent HIV infection in vitro without blocking gp120/CCR5 interaction
Antibodies to the CD4-binding site of HIV-1 gp120 suppress gp120-specific CD4 T cell response while enhancing antibody response
Allosteric induction of the CD4-bound conformation of HIV-1 Gp120
Genome editing of the HIV co-receptors CCR5 and CXCR4 by CRISPR-Cas9 protects CD4+ T cells from HIV-1 infection
Conformational alterations in the CD4 binding cavity of HIV-1 gp120 influencing gp120-CD4 interactions and fusogenicity of HIV-1 envelopes derived from brain and other tissues
Characterization of gp120 and Its Single-chain Derivatives, gp120-CD4 and gp120-M9: Implications for Targeting the CD4i Epitope in HIV-1 Vaccine Design
HIV-1 gp120 receptor on CD4-negative brain cells activates a tyrosine kinase
Recombinant soluble CCR5 AND CXCR4 chemokine receptors as anti-HIV drug targets
IFNα induces CCR5 in CD4+ T cells of HIV patients causing pathogenic elevation
Modellierung der Interaktion des humanen Korezeptor CCR5 mit der V3-Loop des Glycoproteins gp120 aus dem HIV-1
Das "Strukturschalterphänomen" der CD4-Bindungsdomäne des HIV-1-Oberflächenproteins gp120 und dessen exogene Inhibition