The expression of TGF-β1, Smad3, phospho-Smad3 and Smad7 is correlated with the development and invasion of nonfunctioning pituitary adenomas
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1479-5876
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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The expression of TGF-β1, Smad3, phospho-Smad3 and Smad7 is correlated with the development and invasion of nonfunctioning pituitary adenomas ; volume:12 ; number:1 ; day:18 ; month:3 ; year:2014 ; pages:1-8 ; date:12.2014
Journal of translational medicine ; 12, Heft 1 (18.3.2014), 1-8, 12.2014
- Urheber
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Zhenye, Li
- Beteiligte Personen und Organisationen
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Chuzhong, Li
Youtu, Wu
Xiaolei, Lan
Lei, Cao
Lichuan, Hong
Hongyun, Wang
Yonggang, Wu
Fei, Wang
Yazhuo, Zhang
SpringerLink (Online service)
- DOI
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10.1186/1479-5876-12-71
- URN
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urn:nbn:de:1111-2016041448016
- Rechteinformation
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:46 MESZ
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Beteiligte
- Zhenye, Li
- Chuzhong, Li
- Youtu, Wu
- Xiaolei, Lan
- Lei, Cao
- Lichuan, Hong
- Hongyun, Wang
- Yonggang, Wu
- Fei, Wang
- Yazhuo, Zhang
- SpringerLink (Online service)
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Serum TGF-β1 and SMAD3 levels are closely associated with coronary artery disease
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Ovarian cancer-derived TGF-β1 induces cancer-associated adipocytes formation by activating SMAD3/TRIB3 pathway to establish pre-metastatic niche
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Serum TGF-β1 and SMAD3 levels are closely associated with coronary artery disease
Diacerein ameliorates amiodarone-induced pulmonary fibrosis via targeting the TGFβ1/α-SMA/Smad3 pathway
Targeting PSMD14 inhibits melanoma growth through SMAD3 stabilization
All-trans retinoic acid inhibits lipopolysaccharide-induced inflammatory responses in bovine adipocytes via TGFβ1/Smad3 signaling pathway
Downregulation of TGF-β1 suppressed proliferation and increased chemosensitivity of ovarian cancer cells by promoting BRCA1/Smad3 signaling
TGF-β1 induced deficiency of linc00261 promotes epithelial–mesenchymal-transition and stemness of hepatocellular carcinoma via modulating SMAD3
Dopamine and α-synuclein dysfunction in Smad3 null mice
Nuclear NFATc1/Smad3 complexes in Smad4-deficient pancreatic cancer
NSUN5 Facilitates Hepatocellular Carcinoma Progression by Increasing SMAD3 Expression
Smad3 Promotes Cancer‐Associated Fibroblasts Generation via Macrophage–Myofibroblast Transition
Ovarian cancer-derived TGF-β1 induces cancer-associated adipocytes formation by activating SMAD3/TRIB3 pathway to establish pre-metastatic niche