Erratum to: Loss of plakoglobin promotes cell-cell contact, increased invasion and breast cancer cell dissemination in vivo
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1465-542X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Erratum to: Loss of plakoglobin promotes cell-cell contact, increased invasion and breast cancer cell dissemination in vivo ; volume:19 ; number:1 ; day:28 ; month:3 ; year:2017 ; pages:1-2 ; date:12.2017
Breast cancer research ; 19, Heft 1 (28.3.2017), 1-2, 12.2017
- Keyword
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Krebs
Onkologie
- Creator
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Holen, Ingunn
- Contributor
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Whitworth, Jacob
Nutter, Faith
Evans, Alyson
Brown, Hannah K.
Lefley, Diane V.
Barbaric, Ivana
Jones, Mark
Ottewell, Penelope D.
SpringerLink (Online service)
- DOI
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10.1186/s13058-017-0835-4
- URN
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urn:nbn:de:1111-201704157007
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:27 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Holen, Ingunn
- Whitworth, Jacob
- Nutter, Faith
- Evans, Alyson
- Brown, Hannah K.
- Lefley, Diane V.
- Barbaric, Ivana
- Jones, Mark
- Ottewell, Penelope D.
- SpringerLink (Online service)
Other Objects (12)
Loss of plakoglobin promotes decreased cell-cell contact, increased invasion, and breast cancer cell dissemination in vivo
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Platelet and red blood cell interaction – Unraveling of interaction molecules during cell-cell contact
Cell-cell fusion: human multinucleated osteoclasts
Cell–cell crosstalk in the heart
Vinculin in cell–cell and cell–matrix adhesions
PI3K-AKT, JAK2-STAT3 pathways and cell–cell contact regulate maspin subcellular localization
Cell–cell junctional mechanotransduction in endothelial remodeling
Subcellular localization of Type VI secretion system assembly in response to cell–cell contact
Cell-cell contact-induced gene editing/activation in mammalian cells using a synNotch-CRISPR/Cas9 system
Cell-cell contact-driven EphB1 cis- and trans- signalings regulate cancer stem cells enrichment after chemotherapy
Loss of plakoglobin promotes decreased cell-cell contact, increased invasion, and breast cancer cell dissemination in vivo
Cell-Cell Channels
Multiplexing cell‐cell communication
Platelet and red blood cell interaction – Unraveling of interaction molecules during cell-cell contact
Cell-cell fusion: human multinucleated osteoclasts
Cell–cell crosstalk in the heart
Vinculin in cell–cell and cell–matrix adhesions
PI3K-AKT, JAK2-STAT3 pathways and cell–cell contact regulate maspin subcellular localization
Cell–cell junctional mechanotransduction in endothelial remodeling
Subcellular localization of Type VI secretion system assembly in response to cell–cell contact
Cell-cell contact-induced gene editing/activation in mammalian cells using a synNotch-CRISPR/Cas9 system
Cell-cell contact-driven EphB1 cis- and trans- signalings regulate cancer stem cells enrichment after chemotherapy
Loss of plakoglobin promotes decreased cell-cell contact, increased invasion, and breast cancer cell dissemination in vivo
Cell-Cell Channels
Multiplexing cell‐cell communication
Platelet and red blood cell interaction – Unraveling of interaction molecules during cell-cell contact
Cell-cell fusion: human multinucleated osteoclasts
Cell–cell crosstalk in the heart
Vinculin in cell–cell and cell–matrix adhesions
PI3K-AKT, JAK2-STAT3 pathways and cell–cell contact regulate maspin subcellular localization
Cell–cell junctional mechanotransduction in endothelial remodeling
Subcellular localization of Type VI secretion system assembly in response to cell–cell contact
Cell-cell contact-induced gene editing/activation in mammalian cells using a synNotch-CRISPR/Cas9 system