Polymorphisms in RAD51, XRCC2 and XRCC3 genes of the homologous recombination repair in colorectal cancer—a case control study
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1573-4978
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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Polymorphisms in RAD51, XRCC2 and XRCC3 genes of the homologous recombination repair in colorectal cancer—a case control study ; volume:38 ; number:4 ; day:20 ; month:11 ; year:2010 ; pages:2849-2854 ; date:4.2011
Molecular biology reports ; 38, Heft 4 (20.11.2010), 2849-2854, 4.2011
- Urheber
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Krupa, Renata
Sliwinski, Tomasz
Wisniewska-Jarosinska, Maria
Chojnacki, Jan
Wasylecka, Maja
Dziki, Lukasz
Morawiec, Jan
Blasiak, Janusz
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1007/s11033-010-0430-6
- URN
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urn:nbn:de:101:1-2019090114252773393194
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:27 MESZ
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Beteiligte
- Krupa, Renata
- Sliwinski, Tomasz
- Wisniewska-Jarosinska, Maria
- Chojnacki, Jan
- Wasylecka, Maja
- Dziki, Lukasz
- Morawiec, Jan
- Blasiak, Janusz
- SpringerLink (Online service)
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The bromodomain containing protein BRD-9 orchestrates RAD51–RAD54 complex formation and regulates homologous recombination-mediated repair
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Polymerase iota (POLI) confers radioresistance of esophageal squamous cell carcinoma by regulating RAD51 stability and facilitating homologous recombination
CircCDYL2 bolsters radiotherapy resistance in nasopharyngeal carcinoma by promoting RAD51 translation initiation for enhanced homologous recombination repair