Mutational signatures of non‐homologous and polymerase theta‐mediated end‐joining in embryonic stem cells
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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Mutational signatures of non‐homologous and polymerase theta‐mediated end‐joining in embryonic stem cells ; volume:36 ; number:24 ; year:2017 ; pages:3634-3649 ; extent:16
The EMBO journal / European Molecular Biology Organization ; 36, Heft 24 (2017), 3634-3649 (gesamt 16)
- Creator
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Schimmel, Joost
Kool, Hanneke
van Schendel, Robin
Tijsterman, Marcel
- DOI
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10.15252/embj.201796948
- URN
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urn:nbn:de:101:1-2022100307034851635026
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:24 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Schimmel, Joost
- Kool, Hanneke
- van Schendel, Robin
- Tijsterman, Marcel
Other Objects (12)
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PHF6 promotes non‐homologous end joining and G2 checkpoint recovery
Resection-dependent canonical non-homologous end-joining induces genomic rearrangements
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination
Merotelic attachments and non-homologous end joining are the basis of chromosomal instability
SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Impaired non-homologous end joining in human primary alveolar type II cells in emphysema
The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance
Lower genomic stability of induced pluripotent stem cells reflects increased non-homologous end joining
Poly(ADP) ribose polymerase promotes DNA polymerase theta-mediated end joining by activation of end resection
DNA non-homologous end-joining enters the resection arena
PHF6 promotes non‐homologous end joining and G2 checkpoint recovery
Resection-dependent canonical non-homologous end-joining induces genomic rearrangements
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination
Merotelic attachments and non-homologous end joining are the basis of chromosomal instability
SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Impaired non-homologous end joining in human primary alveolar type II cells in emphysema
The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance
Lower genomic stability of induced pluripotent stem cells reflects increased non-homologous end joining
Poly(ADP) ribose polymerase promotes DNA polymerase theta-mediated end joining by activation of end resection
DNA non-homologous end-joining enters the resection arena
PHF6 promotes non‐homologous end joining and G2 checkpoint recovery
Resection-dependent canonical non-homologous end-joining induces genomic rearrangements
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination
Merotelic attachments and non-homologous end joining are the basis of chromosomal instability
SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Impaired non-homologous end joining in human primary alveolar type II cells in emphysema
The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance