Quiescence status of glioblastoma stem-like cells involves remodelling of Ca2+ signalling and mitochondrial shape
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Quiescence status of glioblastoma stem-like cells involves remodelling of Ca2+ signalling and mitochondrial shape ; volume:8 ; number:1 ; day:27 ; month:6 ; year:2018 ; pages:1-12 ; date:12.2018
Scientific reports ; 8, Heft 1 (27.6.2018), 1-12, 12.2018
- Creator
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Aulestia, Francisco J.
- Contributor
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Néant, Isabelle
Dong, Jihu
Haiech, Jacques
Kilhoffer, Marie-Claude
Moreau, Marc
Leclerc, Catherine
SpringerLink (Online service)
- DOI
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10.1038/s41598-018-28157-8
- URN
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urn:nbn:de:101:1-2018083122055089265074
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:37 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Aulestia, Francisco J.
- Néant, Isabelle
- Dong, Jihu
- Haiech, Jacques
- Kilhoffer, Marie-Claude
- Moreau, Marc
- Leclerc, Catherine
- SpringerLink (Online service)
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Proliferation/quiescence: the controversial "aller-retour"
Bradykinin-induced Ca2+ signaling in human subcutaneous fibroblasts involves ATP release via hemichannels leading to P2Y12 receptors activation
Polyamine triggering of exocytosis in Paramecium involves an extracellular Ca2+/(polyvalent cation)-sensing receptor, subplasmalemmal Ca-store mobilization and store-operated Ca2+-influx via unspecific cation channels
Ca2+-dependent endoplasmic reticulum stress correlation with astrogliosis involves upregulation of KCa3.1 and inhibition of AKT/mTOR signaling
Widespread changes in mRNA stability contribute to quiescence-specific gene expression patterns in a fibroblast model of quiescence
Role of FOXO transcription factors in vascular quiescence
Dormant cancer cells: programmed quiescence, senescence, or both?
Pancreatic β-cell mitophagy as an adaptive response to metabolic stress and the underlying mechanism that involves lysosomal Ca2+ release
Nuclear Hsp104 safeguards the dormant translation machinery during quiescence
Immune quiescence: a model of protection against HIV infection
Achieving Quiescence with Fluocinolone Implants
Inequality and quiescence: a continuing conundrum
Proliferation/quiescence: the controversial "aller-retour"
Bradykinin-induced Ca2+ signaling in human subcutaneous fibroblasts involves ATP release via hemichannels leading to P2Y12 receptors activation
Polyamine triggering of exocytosis in Paramecium involves an extracellular Ca2+/(polyvalent cation)-sensing receptor, subplasmalemmal Ca-store mobilization and store-operated Ca2+-influx via unspecific cation channels
Ca2+-dependent endoplasmic reticulum stress correlation with astrogliosis involves upregulation of KCa3.1 and inhibition of AKT/mTOR signaling
Widespread changes in mRNA stability contribute to quiescence-specific gene expression patterns in a fibroblast model of quiescence
Role of FOXO transcription factors in vascular quiescence
Dormant cancer cells: programmed quiescence, senescence, or both?
Pancreatic β-cell mitophagy as an adaptive response to metabolic stress and the underlying mechanism that involves lysosomal Ca2+ release
Nuclear Hsp104 safeguards the dormant translation machinery during quiescence