Expression of K2P5.1 potassium channels on CD4+T lymphocytes correlates with disease activity in rheumatoid arthritis patients
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1478-6354
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Expression of K2P5.1 potassium channels on CD4+T lymphocytes correlates with disease activity in rheumatoid arthritis patients ; volume:13 ; number:1 ; day:11 ; month:2 ; year:2011 ; pages:1-9 ; date:2.2011
Arthritis Research & Therapy ; 13, Heft 1 (11.2.2011), 1-9, 2.2011
- Creator
- Contributor
-
Bobak, Nicole
Feuchtenberger, Martin
Herrmann, Alexander M.
Göbel, Kerstin
Kinne, Raimund W.
HANSEN, ANKER J.
Budde, Thomas
Kleinschnitz, Christoph
Frey, Oliver
Tony, Hans-Peter
Wiendl, Heinz
Meuth, Sven
SpringerLink (Online service)
- DOI
-
10.1186/ar3245
- URN
-
urn:nbn:de:1111-2016050317912
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:47 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Bittner, Stefan
- Bobak, Nicole
- Feuchtenberger, Martin
- Herrmann, Alexander M.
- Göbel, Kerstin
- Kinne, Raimund W.
- HANSEN, ANKER J.
- Budde, Thomas
- Kleinschnitz, Christoph
- Frey, Oliver
- Tony, Hans-Peter
- Wiendl, Heinz
- Meuth, Sven
- SpringerLink (Online service)
Other Objects (12)
Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)
Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)
Evans syndrome associated with sterile inflammation of the central nervous system: a case report
Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter
Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter
CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level
Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation
Wave-dynamical properties of dielectric resonators investigated with microwaves
Wave-dynamical properties of dielectric resonators investigated with microwaves
Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration
Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms
Immunregulatorische Eigenschaften des Muskels : Implikationen für die Pathogenese entzündlicher Myopathien
Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)
Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)
Evans syndrome associated with sterile inflammation of the central nervous system: a case report
Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter
Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter
CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level
Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation
Wave-dynamical properties of dielectric resonators investigated with microwaves
Wave-dynamical properties of dielectric resonators investigated with microwaves
Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration
Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms
Immunregulatorische Eigenschaften des Muskels : Implikationen für die Pathogenese entzündlicher Myopathien
Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)
Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)
Evans syndrome associated with sterile inflammation of the central nervous system: a case report
Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter
Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter
CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level
Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation
Wave-dynamical properties of dielectric resonators investigated with microwaves
Wave-dynamical properties of dielectric resonators investigated with microwaves
Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration
Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms