TRPC3 overexpression promotes angiotensin II-induced cardiac dysfunction

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2050-6511

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: TRPC3 overexpression promotes angiotensin II-induced cardiac dysfunction ; volume:13 ; number:1 ; day:17 ; month:9 ; year:2012 ; pages:1-2 ; date:9.2012
BMC pharmacology & toxicology ; 13, Heft 1 (17.9.2012), 1-2, 9.2012

Creator: Doleschal, Bernhard

Contributor: Wolf, Stefan
Huber, Marie-Sophie
Wölkart, Gerald
Antoons, Gudrun
Groschner, Klaus
SpringerLink (Online service)

DOI: 10.1186/2050-6511-13-S1-A83

URN: urn:nbn:de:1111-2016073153963

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:59 AM CEST

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Doleschal, Bernhard
Wolf, Stefan
Huber, Marie-Sophie
Wölkart, Gerald
Antoons, Gudrun
Groschner, Klaus
SpringerLink (Online service)

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TRPC3 overexpression promotes angiotensin II-induced cardiac dysfunction

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Other Objects (12)

JDP2 overexpression provokes cardiac dysfunction in mice

Overexpression of the human angiotensin II type I receptor in neonatal cardiac myocytes directly promotes hypertrophic responses to angiotensin II in vitro

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Human paraoxonase gene cluster overexpression alleviates angiotensin II-induced cardiac hypertrophy in mice

Kaempferol Alleviates Angiotensin II-Induced Cardiac Dysfunction and Interstitial Fibrosis in Mice

Angiotensin type 1a receptor-deficient mice develop diabetes-induced cardiac dysfunction, which is prevented by renin-angiotensin system inhibitors

IL-37 overexpression promotes endometrial regenerative cell-mediated inhibition of cardiac allograft rejection

Cardiomyocyte-specific RXFP1 overexpression protects against pressure overload-induced cardiac dysfunction independently of Relaxin

Cardiac-specific catalase overexpression rescues anthrax lethal toxin-induced cardiac contractile dysfunction: role of oxidative stress and autophagy

Liver-specific NG37 overexpression leads to diet-dependent fatty liver disease accompanied by cardiac dysfunction

Overexpression of CPEB3 leads to astrocyte dysfunction

Overexpression of CPEB3 leads to astrocyte dysfunction

JDP2 overexpression provokes cardiac dysfunction in mice

Overexpression of the human angiotensin II type I receptor in neonatal cardiac myocytes directly promotes hypertrophic responses to angiotensin II in vitro

Cardiac-specific overexpression of Ndufs1 ameliorates cardiac dysfunction after myocardial infarction by alleviating mitochondrial dysfunction and apoptosis

Human paraoxonase gene cluster overexpression alleviates angiotensin II-induced cardiac hypertrophy in mice

Kaempferol Alleviates Angiotensin II-Induced Cardiac Dysfunction and Interstitial Fibrosis in Mice

Angiotensin type 1a receptor-deficient mice develop diabetes-induced cardiac dysfunction, which is prevented by renin-angiotensin system inhibitors

IL-37 overexpression promotes endometrial regenerative cell-mediated inhibition of cardiac allograft rejection

Cardiomyocyte-specific RXFP1 overexpression protects against pressure overload-induced cardiac dysfunction independently of Relaxin

Cardiac-specific catalase overexpression rescues anthrax lethal toxin-induced cardiac contractile dysfunction: role of oxidative stress and autophagy

Liver-specific NG37 overexpression leads to diet-dependent fatty liver disease accompanied by cardiac dysfunction

Overexpression of CPEB3 leads to astrocyte dysfunction

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Overexpression of the human angiotensin II type I receptor in neonatal cardiac myocytes directly promotes hypertrophic responses to angiotensin II in vitro

Cardiac-specific overexpression of Ndufs1 ameliorates cardiac dysfunction after myocardial infarction by alleviating mitochondrial dysfunction and apoptosis

Human paraoxonase gene cluster overexpression alleviates angiotensin II-induced cardiac hypertrophy in mice

Kaempferol Alleviates Angiotensin II-Induced Cardiac Dysfunction and Interstitial Fibrosis in Mice

Angiotensin type 1a receptor-deficient mice develop diabetes-induced cardiac dysfunction, which is prevented by renin-angiotensin system inhibitors

IL-37 overexpression promotes endometrial regenerative cell-mediated inhibition of cardiac allograft rejection

Cardiomyocyte-specific RXFP1 overexpression protects against pressure overload-induced cardiac dysfunction independently of Relaxin

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Liver-specific NG37 overexpression leads to diet-dependent fatty liver disease accompanied by cardiac dysfunction

Overexpression of CPEB3 leads to astrocyte dysfunction

Overexpression of CPEB3 leads to astrocyte dysfunction

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